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Founded in:
2003-11-12 -
Country:
China -
Address:
No. 5, Central Road, Ulanhot Economic and Technological Development Zone, Inner Mongolia Autonomous Region -
Tax NO.:
91150000701336455A -
Registered Funds:
140 million yuan -
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Diammonium glycyrrhizinate |
This product has strong anti-inflammatory, liver cell membrane protection and liver function improvement effects. Pharmacological experiments have shown that oral administration of this product to mice can reduce the increase of serum alanine aminotransferase and aspartate aminotransferase caused by carbon tetrachloride, thioacetamide and D-galactosamine. It can also significantly reduce the morphological damage of D-galactosamine to the liver and improve the chronic damage of immune factors to the liver morphology.
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This product has strong anti-inflammatory, liver cell membrane protection and liver function improvement effects. Pharmacological experiments have shown that oral administration of this product to mice can reduce the increase of serum alanine aminotransferase and aspartate aminotransferase caused by carbon tetrachloride, thioacetamide and D-galactosamine. It can also significantly reduce the morphological damage of D-galactosamine to the liver and improve the chronic damage of immune factors to the liver morphology. |
79165-06-3 | 35 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Propyl gallate |
1. It can obviously inhibit the synthesis of thromboxane A2 (TXA2), counteract the platelet aggregation caused by arachidonic acid (AA), and has a stronger and faster antiplatelet aggregation effect than aspirin (ASP). 2. It can enhance the fibrinolytic activity and promote the dissolution of thrombus. 3. It can reduce the specific viscosity of whole blood and plasma, and accelerate the electrophoresis of red blood cells. 4. It can relax the vascular smooth muscle, dilate the artery, and increase the blood flow of the coronary artery and cerebral artery. 5. It can improve the ability to resist hypoxia. 6. It can strongly scavenge free radicals. 7. It can inhibit the inflammatory exudation of capillaries.
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1. It can obviously inhibit the synthesis of thromboxane A2 (TXA2), counteract the platelet aggregation caused by arachidonic acid (AA), and has a stronger and faster antiplatelet aggregation effect than aspirin (ASP). 2. It can enhance the fibrinolytic activity and promote the dissolution of thrombus. 3. It can reduce the specific viscosity of whole blood and plasma, and accelerate the electrophoresis of red blood cells. 4. It can relax the vascular smooth muscle, dilate the artery, and increase the blood flow of the coronary artery and cerebral artery. 5. It can improve the ability to resist hypoxia. 6. It can strongly scavenge free radicals. 7. It can inhibit the inflammatory exudation of capillaries. |
121-79-9 | 10 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Piracetam |
Extract from the above information
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Extract from the above information |
7491-74-9 | 24 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| (+/-)-Verapamil hydrochloride |
1. Calcium ion antagonist, regulates calcium ion influx on the membrane of myocardial conduction cells, myocardial contractile cells and arterial smooth muscle cells, without changing serum calcium concentration; 2. Dilates the main coronary arteries and arterioles, antagonizes coronary artery spasm, increases myocardial oxygen delivery, reduces peripheral resistance, and reduces myocardial oxygen consumption; 3. Prolongs the effective refractory period of the atrioventricular node, slows conduction, and reduces the ventricular rate of patients with chronic atrial fibrillation and atrial flutter; 4. Reduces systemic vascular resistance to produce a hypotensive effect, generally without causing postural hypotension or reflex tachycardia; 5. Reduces afterload, inhibits myocardial contraction, and improves left ventricular diastolic function; 6. Animal experiments show that the local anesthetic effect is 1.6 times that of procaine equimolar.
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1. Calcium ion antagonist, regulates calcium ion influx on the membrane of myocardial conduction cells, myocardial contractile cells and arterial smooth muscle cells, without changing serum calcium concentration; 2. Dilates the main coronary arteries and arterioles, antagonizes coronary artery spasm, increases myocardial oxygen delivery, reduces peripheral resistance, and reduces myocardial oxygen consumption; 3. Prolongs the effective refractory period of the atrioventricular node, slows conduction, and reduces the ventricular rate of patients with chronic atrial fibrillation and atrial flutter; 4. Reduces systemic vascular resistance to produce a hypotensive effect, generally without causing postural hypotension or reflex tachycardia; 5. Reduces afterload, inhibits myocardial contraction, and improves left ventricular diastolic function; 6. Animal experiments show that the local anesthetic effect is 1.6 times that of procaine equimolar. |
152-11-4 | 13 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Potassium L-aspartate |
|
14007-45-5 | 11 |