This product is a kind of mucopolysaccharide hydrolase widely distributed in organisms. It can liquefy the insoluble polysaccharides in the cell walls of Gram-positive bacteria and hydrolyze them into soluble mucopeptides. It is a natural anti-infective substance with bactericidal effects. It has antibacterial, antiviral, anti-inflammatory effects, can enhance the efficacy of antibiotics and accelerate tissue recovery.

1. It is a non-selective competitive inhibitor of adrenergic β-receptor blockers, blocking β1 and β2 receptors on the heart, reducing the contractile force and contractile speed of the heart, while inhibiting the contraction of vascular smooth muscle, reducing myocardial oxygen consumption, and restoring the oxygen supply and demand relationship of ischemic myocardium to a low level. It is used to treat angina pectoris. 2. It inhibits the adrenergic excitement of cardiac pacemaker potentials and is used to treat arrhythmias. 3. It can inhibit the release of renin and reduce cardiac output through central and adrenergic neuron blockade, and is used to treat hypertension. 4. It competitively antagonizes the effects of isoproterenol and norepinephrine, blocks β2 receptors, and reduces plasma-related activity.

This product is an analogue of diphenhydramine, which mainly suppresses cough center and relieves cough. It also has a weak antihistamine effect and has no dependence or tolerance. It takes effect 20-30 minutes after taking it and the effect lasts for 3-4 hours.

Promotes the physiological release of acetylcholine in the myenteric plexus; does not stimulate muscarinic and nicotinic receptors, nor inhibit the activity of acetylcholinesterase; has no dopamine receptor blocking effect at therapeutic doses; is mainly distributed in the stomach and intestinal tissues. In humans: enhances esophageal peristalsis and lower esophageal sphincter tension, prevents gastric contents from refluxing into the esophagus, and improves esophageal clearance; increases gastric and duodenal contractility and coordination of the antrum-duodenum, reduces duodenal-gastric reflux, improves gastric and duodenal emptying; strengthens intestinal motility and promotes transit of the small and large intestines. Due to the lack of cholinergic effect, it does not increase the basal and pentagastrin-induced gastric acid secretion; due to the relative lack of dopamine antagonist properties, it has almost no effect on plasma prolactin levels; does not affect psychomotor function, blood pressure, respiratory rate, body temperature, body weight and anticoagulation function; pharmacological action begins 30 to 60 minutes after oral administration.