This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects

This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects

This product can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects

It inhibits the synthesis and release of prostaglandins in the hypothalamus, restores the normal responsiveness of the sensory neurons in the temperature regulation center, and has an antipyretic effect; it has an analgesic effect by inhibiting the synthesis of prostaglandins, etc.; it inhibits the synthesis and release of prostaglandins in the local tissue of inflammation, stabilizes lysosomal enzymes, and affects the phagocytic function of phagocytes, thus playing an anti-inflammatory role

It is a peripheral analgesic that inhibits the synthesis and release of prostaglandins (PGE1) in the hypothalamic temperature regulation center, causing peripheral vasodilation and sweating to achieve an antipyretic effect. It can also inhibit the effects of PGE1, bradykinin and histamine, increase the pain threshold and produce analgesic effects.

It is a central nervous system stimulant that can excite the cerebral cortex, increase sensitivity to the outside world, and constrict cerebral blood vessels, thus enhancing the headache relief effects of the first two drugs.

It has a strong histamine H1 receptor blocking effect, can alleviate other respiratory symptoms caused by allergies, and also has a mild inhibitory effect on the central nervous system

Antiepileptic drugs and antiarrhythmic drugs increase the outflow of sodium ions from cells, reduce the inflow of sodium ions, stabilize the nerve cell membrane, increase the excitation threshold, and reduce the spread of high-frequency discharges in the lesions; shorten the action potential interval and the effective refractory period, inhibit the influx of calcium ions, reduce myocardial automaticity, and have an inhibitory effect on ectopic rhythm points in the atria and ventricles; have anti-neuralgia and skeletal muscle relaxation effects; can inhibit the synthesis (or) secretion of collagenase by skin fibroblasts, accelerate vitamin D metabolism, cause lymphadenopathy, have anti-folate effects, have an inhibitory effect on the hematopoietic system, can cause allergic reactions, have enzyme induction effects, and intravenous administration can dilate peripheral blood vessels.

This product is a nitroimidazole derivative that can inhibit the redox reaction of amoeba and break the nitrogen chain of the protozoa. In vitro tests have shown that when the drug concentration is 1~2mg/L, the histolytica amoeba can undergo morphological changes in 6~20 hours and all are killed within 24 hours. When the concentration is 0.2mg/L, the histolytica amoeba can be killed within 72 hours. This product has a strong effect of killing trichomonas, and its mechanism is unknown. Metronidazole has a killing effect on anaerobic microorganisms, and the metabolites generated when it is reduced in the human body also have anti-anaerobic effects, inhibiting the synthesis of bacterial deoxyribonucleic acid, thereby interfering with the growth and reproduction of bacteria, and ultimately causing bacterial death. It has a carcinogenic effect on some animals.