This product is one of the ingredients of the Chinese medicine bezoar. As an endogenous amino acid in the human body, this product is a central inhibitory transmitter that can regulate the excitability of nerve tissue and body temperature. Therefore, it has antipyretic, sedative, analgesic, anti-inflammatory, anti-rheumatic, and anticonvulsant effects. In addition, it can improve the body's nonspecific immune function.

Participate in some metabolic reactions in the body. It can fight against the peroxidation of free radicals, resist aging, protect the skin, enhance ovarian function, and prevent habitual abortion.

This product participates in some metabolic reactions in the body. It can fight against the peroxidation of free radicals, resist aging, protect the skin, enhance ovarian function, and prevent habitual abortion.

Rifampicin is a semi-synthetic broad-spectrum antibacterial drug of the rifamycin class, which has antibacterial activity against a variety of pathogenic microorganisms. The drug has a significant bactericidal effect on Mycobacterium tuberculosis and some non-tuberculous mycobacteria (including Mycobacterium leprae, etc.) both inside and outside the host cells. Rifampicin has a good antibacterial effect on aerobic Gram-positive bacteria, including enzyme-producing strains of Staphylococcus aureus and methicillin-resistant strains, Streptococcus pneumoniae, other Streptococcus, Enterococcus, Listeria, Bacillus anthracis, Clostridium perfringens, Corynebacterium diphtheriae, anaerobic cocci, etc. It also has a high degree of antibacterial activity against aerobic Gram-negative bacteria such as Neisseria meningitidis, Haemophilus influenzae, and Neisseria gonorrhoeae. Rifampicin also has a good effect on Legionella, and has an inhibitory effect on pathogens such as Chlamydia trachomatis, lymphogranuloma venereum, and psittacosis.

This product is an intermediate in the synthesis of Schisandrae Chinensis A, which can reduce liver damage and alanine aminotransferase (ALT) elevation caused by carbon tetrachloride, inhibit liver microsomal lipid peroxidation and metabolic conversion to carbon monoxide caused by carbon tetrachloride, reduce the consumption of reduced coenzyme II and oxygen in the metabolic process, and protect the structure and function of liver cell biomembrane; it can reduce the increase of liver ALT induced by prednisone, promote liver regeneration in mice with partial hepatectomy; it has a significant induction effect on cytochrome P450 enzyme activity, and enhances the detoxification ability of carbon tetrachloride and certain carcinogens; it has an effect on improving protein metabolism in some hepatitis patients, increasing albumin and reducing globulin. It has no negative conversion effect on HbsAg and HbeAg, and cannot shrink enlarged liver and spleen.

It can be converted into PEG2, PGI2, and PGF2a, and has obvious lipid-lowering and anti-platelet aggregation effects; it has obvious cholesterol-lowering effects, and its efficacy is 163 times that of linoleic acid; it can participate in the synthesis and balance of prostaglandins; it can significantly inhibit platelet aggregation and the synthesis of thromboxane A2 (TXA2) by platelets, thereby changing the TXA2/PGI2 ratio, and has the effect of preventing and treating thrombotic cardiovascular diseases; it has a stimulating effect on brown adipose tissue, promotes the activity of brown fatty acid mitochondria, consumes excess calories and achieves a certain weight loss effect.