Methylcobalamin is an endogenous coenzyme B12 that participates in the one-carbon unit cycle and plays an important role in the transmethylation reaction of methionine synthesis from homocysteine. Animal experiments show that this product is easier to enter neuronal organelles than cyanocobalamin, participates in the synthesis of thymidine nucleoside in brain cells and spinal cord neurons, promotes the utilization of folic acid and nucleic acid metabolism, and has a stronger effect on promoting nucleic acid and protein synthesis than cyanocobalamin; it can promote axonal transport function and axonal regeneration, and normalize the transport of axonal skeleton proteins in the sciatic nerve of diabetic rats induced by streptozotocin; it has an inhibitory effect on drug-induced neurodegeneration, such as axonal degeneration in mice caused by adriamycin, acrylamide, and vincristine, and neurological diseases in spontaneously hypertensive rats; it can restore delayed synaptic transmission and neurotransmitter reduction to normal, and restore acetylcholine in the brain of rats fed with choline-deficient feed to normal levels by increasing nerve fiber excitability and restoring ultimate plate potential induction. In vitro studies have shown that methylcobalamin can promote the synthesis of lecithin and neuronal myelination in cultured rat tissues.

Smooth muscle relaxant. It has the function of inhibiting adenylate cyclase and phosphodiesterase, antagonizing calcium ions, and has a weak antimuscarinic effect. It has a selective antispasmodic effect on the smooth muscles of the urogenital system, can relieve the spasm of the smooth muscles of the urogenital system, relax the muscles, and eliminate frequent urination, urgency, urinary incontinence, and lower abdominal pain caused by urethral bladder smooth muscle spasm.

This product is a mucolytic agent that can increase the secretion of serous glands in the respiratory mucosa and reduce the secretion of mucous glands, thereby reducing sputum viscosity, promoting the secretion of pulmonary surfactant, increasing bronchial ciliary movement, and making sputum easier to cough up.

This product is an H2 receptor inhibitor, which has the effect of inhibiting gastric acid secretion. It is rapidly absorbed through the gastrointestinal tract after oral administration.

This product is an H2 receptor inhibitor, which has the effect of inhibiting gastric acid secretion. It is rapidly absorbed through the gastrointestinal tract after oral administration.

This product is an H2 receptor inhibitor, which has the effect of inhibiting gastric acid secretion. It is rapidly absorbed through the gastrointestinal tract after oral administration.

This product is an H2 receptor inhibitor, which has the effect of inhibiting gastric acid secretion. It is rapidly absorbed through the gastrointestinal tract after oral administration.

1. Blocking effect on sympathetic nerve receptors: It selectively blocks alpha 1 receptors, and its effect is 0.5-22 times stronger than prazosin hydrochloride and 45-140 times stronger than phentolamine mesylate; its affinity for alpha 1 receptors is 5400-24000 times stronger than alpha 2 receptors. 2. Effects on the urethra, bladder and prostate: As a specific antagonist of alpha 1 receptor subtype alpha 1A, it has a highly selective blocking effect on the urethra, bladder neck and prostate smooth muscle, relaxes the smooth muscle, reduces urethral pressure, and its ability to inhibit the increase of urethral pressure is 13 times that of inhibiting the increase of vasodilation pressure. 3. Effect on improving urination disorders: It can reduce the prostate pressure in the urethral pressure curve, and has no effect on rhythmic bladder contraction and bladder pressure curve.