It selectively inhibits succinate dehydrogenase in the muscles of the worm, affects the anaerobic metabolism of the muscles of the worm, and reduces energy production; it depolarizes the nerve muscles, causing continuous muscle contraction and paralysis; its cholinergic effect is beneficial to the excretion of the worm; it may have an inhibitory effect on the microtubule structure of the worm; it has immune regulation and immune excitation functions.

This product has good antibacterial effect on superficial fungi such as Trichophyton, Microsporum, Epidermophyton, etc. It has no antibacterial effect on Candida, Cryptococcus, Histoplasma, Sporothrix, Blastomyces, Coccidioides, etc. This drug inhibits the growth of fungi by interfering with the synthesis of fungal nucleic acids.

Quinolone antibiotics inhibit bacterial DNA replication by inhibiting bacterial DNA gyrase activity. They have good antibacterial effects on most Enterobacteriaceae, some Staphylococci, Streptococcus pneumoniae, Haemophilus influenzae, Pseudomonas aeruginosa, Neisseria gonorrhoeae, Chlamydia, etc., with a broad antibacterial spectrum and strong antibacterial effects.

Macrolide antibiotics have inhibitory effects on Gram-positive bacteria such as Staphylococcus aureus, Streptococcus, and Pneumococcus, as well as some Gram-negative bacteria such as Haemophilus influenzae, Bordetella pertussis, Neisseria gonorrhoeae, Legionella pneumophila, and some anaerobic bacteria such as Bacteroides fragilis, Streptococcus peptostreptococcus, and Propionibacterium acnes. In addition, they also have inhibitory effects on mycoplasmas. In vivo, the antibacterial activity against some bacteria such as Staphylococcus aureus, Streptococcus, and Haemophilus influenzae is stronger than that of erythromycin. The antibacterial effect is produced by hindering the association of the 50S subunit of the nucleoprotein and inhibiting protein synthesis.

It is oxidized in the body to phenobarbital and phenethyl malonamide, which have anti-epileptic activity. Its anti-epileptic effect is similar to that of phenobarbital, but its intensity and toxicity are lower. It is rapidly and completely absorbed orally, reaching the peak plasma concentration in about 3 hours. The half-life is about 8 hours, and the plasma protein binding rate is 20%. It can easily pass through the blood-brain barrier, the placenta, and appear in breast milk. It mimics the effect of gamma-aminobutyric acid (GABA). At therapeutic concentrations, it can reduce the excitatory effect of glutamate, strengthen the inhibitory effect of gamma-aminobutyric acid, inhibit monosynaptic and polysynaptic transmission in the central nervous system, lead to a decrease in the excitability of the entire nerve cell, and increase the electrical stimulation threshold of the motor cortex. It can increase the threshold of seizures and inhibit the spread of discharges from epileptic foci.