Extract from the above information

Within the therapeutic dose range, it has both (1) and non-selective (2) receptor blocking effects, and has no intrinsic sympathomimetic activity. It blocks the (1) receptors on the postsynaptic membrane, thereby dilating blood vessels and reducing peripheral vascular resistance; it blocks the (2) receptors, inhibits the secretion of renin by the kidneys, blocks the renin-angiotensin-aldosterone system, and produces a hypotensive effect. Carvedilol lowers blood pressure rapidly and can maintain its hypotensive effect for a long time. It has no effect on left ventricular ejection fraction, cardiac function, renal function, renal blood perfusion, peripheral blood flow, plasma electrolytes and blood lipid levels, does not affect heart rate or slightly slows it down, and rarely produces water and sodium retention.

Dilate blood vessels, inhibit platelet aggregation; stabilize liver cell membranes and improve liver function

After entering the blood, most of this product binds to plasma proteins and accumulates in tissues, with the largest accumulation in skeletal muscle. When the concentration in plasma decreases, it is released again. Most of this product is hydrolyzed and converted by plasma cholinesterase, metabolized by the liver into p-aminobenzoic acid and dimethylaminoethanol, and then degraded or combined and excreted in the urine.

This product is a broad-spectrum antifungal drug with good antibacterial effect on many fungi, especially Candida albicans. Its mechanism of action is to inhibit the synthesis of fungal cell membranes and affect their metabolic process.