It has high antibacterial activity against common aerobic Gram-positive bacteria, such as Staphylococcus aureus (including those resistant to penicillin G), Staphylococcus epidermidis, beta-hemolytic Streptococcus, viridans Streptococcus and Streptococcus pneumoniae. It has good antibacterial effect on anaerobic bacteria, including Clostridium diphtheriae, and Clostridium perfringens. It has no activity against Gram-negative bacteria such as Enterococcus, meningococci, Neisseria gonorrhoeae, and Haemophilus influenzae, as well as fungi. There is no cross-resistance with penicillin, chloramphenicol, cephalosporins and tetracyclines, and there is partial cross-resistance with macrolides. It acts on the 50S subunit of the ribosome of sensitive bacteria, preventing the extension of the peptide chain, thereby inhibiting the protein synthesis of bacterial cells. It is generally an antibacterial agent, but at high concentrations, it also has a bactericidal effect on certain bacteria.

It prompts the body to increase the plasma concentration of ACTH and cortisone, promotes the blood vessel wall to increase the secretion of PGF2alpha, scavenges free radicals in the body, has anti-inflammatory and anti-exudation effects, increases venous tension, accelerates venous blood flow, promotes lymphatic return, improves blood circulation and microcirculation, and protects the blood vessel wall.

It prompts the body to increase the plasma concentration of ACTH and cortisone, promotes the blood vessel wall to increase the secretion of PGF2alpha, scavenges free radicals in the body, has anti-inflammatory and anti-exudation effects, increases venous tension, accelerates venous blood flow, promotes lymphatic return, improves blood circulation and microcirculation, and protects the blood vessel wall.

1. It has a strong antibacterial effect on most anaerobic bacteria, but has no effect on aerobic bacteria and facultative anaerobic bacteria. The antibacterial spectrum includes Bacteroides fragilis and other Bacteroides, Fusobacterium, Clostridium perfringens, Eubacterium, Veillonella, Peptococcus and Peptostreptococcus, etc. Actinomycetes, Lactobacillus and Propionibacterium are resistant to this product. Its bactericidal concentration is slightly higher than the inhibitory concentration. 2. It can inhibit the redox reaction of amoeba and break the nitrogen chain of protozoa. In vitro tests have shown that when the drug concentration is 1-2 mg/L, the morphology of Amoeba histolytica can change in 6-20 hours and all are killed within 24 hours. When the concentration is 0.2 mg/L, Amoeba histolytica can be killed within 72 hours. 3. It has a strong effect of killing trichomonas, and its mechanism is unknown. It has a carcinogenic effect on some animals.

Potassium is the main cation in cells, with a concentration of 150-160 mmol/L, while the main cation outside cells is sodium ions, and the serum potassium concentration is only 3.5-5.0 mmol/L. The body mainly relies on Na+-K+ ATPase on the cell membrane to maintain the difference in K+ and Na+ concentrations inside and outside cells. The acid-base balance in the body has an impact on potassium metabolism. For example, when acidosis occurs, H+ enters the cell. In order to maintain the potential difference inside and outside the cell, K+ is released outside the cell, causing or aggravating hyperkalemia. Metabolic disorders can also affect acid-base balance. The normal potassium ion concentration and concentration difference inside and outside cells are closely related to certain functions of cells, such as carbohydrate metabolism, glycogen storage and protein metabolism, excitability and conductivity of nerves and muscles including myocardium, etc.

It combines with pyrophosphate in the body to form a coenzyme, participates in the oxidative decarboxylation reaction of pyruvate and α-ketoglutaric acid in sugar metabolism, and is necessary for sugar metabolism. When it is deficient, oxidation is blocked to form pyruvate, lactic acid accumulates, and affects the body's energy supply. Its symptoms are mainly manifested in the nervous and cardiovascular systems, with multiple peripheral neuritis affecting both sensory nerves and motor nerves, manifested as paresthesia, neuralgia, limb weakness, muscle soreness and atrophy. In terms of cardiovascular, due to the increase of acetone and lactic acid in the blood, the small arteries are dilated, the diastolic pressure is reduced, and the myocardial metabolism is disordered, which is prone to symptoms of heart failure such as palpitations, shortness of breath, chest tightness, cardiac hypertrophy, liver and lung congestion and peripheral edema. In terms of the digestive tract, it is manifested as weakness and weight loss caused by decreased appetite.