This product belongs to the macrolide antibiotics. It has antibacterial activity against Staphylococcus (except methicillin-resistant strains), all groups of streptococci and Gram-positive bacilli. Neisseria, Haemophilus influenzae, Bordetella pertussis, etc. are also sensitive to this product. This product also has antibacterial effects on various anaerobic bacteria except Bacteroides fragilis and Fusobacterium. It also has inhibitory effects on Legionella, Campylobacter fetus, certain spirochetes, Mycoplasma pneumoniae, Rickettsia and Chlamydia. This product is an antibacterial agent, but it also has a bactericidal effect on certain bacteria at high concentrations. It can penetrate the bacterial cell membrane and reversibly bind to the 50S subunit of the bacterial ribosome near the donor site ("P" site), blocking the transfer RNA (t-RNA) from binding to the "P" site, and also blocking the self-acceptor site ("A" site) of the polypeptide chain.

Extract from the above information

1. Regulating gastric motility: can reduce the amplitude of gastric autonomic motility or make it regular; 2. Inducing propulsive motility of the digestive tract system: can induce physiological propulsive motility of the digestive tract in the adult digestive system; 3. Improving gastric emptying function: can improve gastric emptying function in patients with chronic gastritis; 4. Regulating intestinal motility: can regulate muscle tension in colon specimens and inhibit hypermotility of the large intestine; 5. Regulating lower esophageal sphincter pressure (LESP): can reduce the increase in internal pressure caused by tetragastrin load and recover the decrease in internal pressure caused by secretin; 6. Direct effect on digestive tract smooth muscle: under various conditions, it still has a direct effect on digestive tract smooth muscle, and does not competitively inhibit the contraction caused by acetylcholine; 7. Peripheral antiemetic effect: can prolong the time required for vomiting induced by copper sulfate.

It can inhibit the synthesis of prostaglandins and has antipyretic and analgesic effects

Can enhance the body's resistance

By protecting the energy metabolism of cells under hypoxia or ischemia, preventing the decrease of intracellular ATP levels, it ensures the normal function of ion pumps and the normal operation of transmembrane sodium-potassium flow, and maintains the stability of the intracellular environment. Animal studies have shown that it can help maintain the energy metabolism of the heart and nerve sensory organs under ischemia and hypoxia, reduce intracellular acidosis and changes in transmembrane ion flow caused by ischemia, reduce the movement and infiltration of multinuclear neutrophils during ischemia and myocardial reperfusion, reduce the scope of experimental infarction, and have no significant effect on hemodynamics. Human studies have shown that it can increase coronary blood flow reserve, delay the occurrence of exercise-induced ischemia, limit rapid fluctuations in blood pressure without significant changes in heart rate, significantly reduce the frequency of angina attacks, and significantly reduce the use of glyceryl trinitrate.