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Founded in:
2001-01-17 -
Country:
China -
Address:
No. 17, Zhonghe Street, Beijing Economic and Technological Development Zone, Beijing -
Tax NO.:
911103028011490830 -
Registered Funds:
$9.8 million -
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Galantamine Hydrobromide |
Anticholinesterase drugs can pass through the blood-brain barrier, promote the acquisition of passive avoidance operation in mice, and significantly improve the memory consolidation disorders caused by scopolamine and sodium nitrite; they have a good promoting effect on the learning and memory reproduction of light and dark discrimination operation in rats.
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Anticholinesterase drugs can pass through the blood-brain barrier, promote the acquisition of passive avoidance operation in mice, and significantly improve the memory consolidation disorders caused by scopolamine and sodium nitrite; they have a good promoting effect on the learning and memory reproduction of light and dark discrimination operation in rats. |
1953-04-4 | 16 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Docetaxel |
Docetaxel is a paclitaxel anti-tumor drug that exerts its anti-tumor effect by interfering with the microtubule network necessary for cell mitosis and interphase cell function. Docetaxel can bind to free tubulin, promote tubulin assembly into stable microtubules, and inhibit its depolymerization, resulting in the production of microtubule bundles that have lost their normal functions and the fixation of microtubules, thereby inhibiting cell mitosis. The binding of docetaxel to microtubules does not change the number of protofilaments. This is different from most spindle toxic drugs currently used in clinical practice.
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Docetaxel is a paclitaxel anti-tumor drug that exerts its anti-tumor effect by interfering with the microtubule network necessary for cell mitosis and interphase cell function. Docetaxel can bind to free tubulin, promote tubulin assembly into stable microtubules, and inhibit its depolymerization, resulting in the production of microtubule bundles that have lost their normal functions and the fixation of microtubules, thereby inhibiting cell mitosis. The binding of docetaxel to microtubules does not change the number of protofilaments. This is different from most spindle toxic drugs currently used in clinical practice. |
114977-28-5 | 49 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| CeftazidiMe |
Vitamin B complex participates in the body's metabolism process. It is a necessary coenzyme for many metabolic links in the body and an important coenzyme for tissue respiration. Vitamin B1 is an important component of coenzymes required for carbohydrate metabolism. Vitamin B2 is an important coenzyme component required for tissue respiration. Vitamin B6 is a cofactor for many enzymes and participates in the metabolism of amino acids and fats. Nicotinamide is a component of coenzymes I and II, which is necessary for lipid metabolism, oxidation of tissue respiration and glycogenolysis. Sodium pantothenate is a precursor of coenzyme A and participates in the metabolism of carbohydrates, fats and proteins.
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Vitamin B complex participates in the body's metabolism process. It is a necessary coenzyme for many metabolic links in the body and an important coenzyme for tissue respiration. Vitamin B1 is an important component of coenzymes required for carbohydrate metabolism. Vitamin B2 is an important coenzyme component required for tissue respiration. Vitamin B6 is a cofactor for many enzymes and participates in the metabolism of amino acids and fats. Nicotinamide is a component of coenzymes I and II, which is necessary for lipid metabolism, oxidation of tissue respiration and glycogenolysis. Sodium pantothenate is a precursor of coenzyme A and participates in the metabolism of carbohydrates, fats and proteins. |
72558-82-8 | 27 | |
| Multivitamin B |
It participates in the body's metabolic process, is a necessary coenzyme for various metabolic links in the body, and provides an important coenzyme for tissue respiration.
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It participates in the body's metabolic process, is a necessary coenzyme for various metabolic links in the body, and provides an important coenzyme for tissue respiration. |
0 | ||
| Thiamine chloride |
An important component of coenzymes required for carbohydrate metabolism.
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An important component of coenzymes required for carbohydrate metabolism. |
59-43-8 | 9 | |
| Riboflavin |
An important coenzyme component required for tissue respiration.
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An important coenzyme component required for tissue respiration. |
83-88-5 | 19 | |
| Vitamin B6 |
A cofactor for many enzymes, involved in the metabolism of amino acids and fats.
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A cofactor for many enzymes, involved in the metabolism of amino acids and fats. |
8059-24-3 | 13 | |
| Nicotinamide |
A component of coenzymes I and II, it is necessary for lipid metabolism, oxidation of tissue respiration and glycogenolysis.
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A component of coenzymes I and II, it is necessary for lipid metabolism, oxidation of tissue respiration and glycogenolysis. |
98-92-0 | 12 | |
| Sodium D-pantothenate |
A precursor of coenzyme A, involved in the metabolism of carbohydrates, fats and proteins.
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A precursor of coenzyme A, involved in the metabolism of carbohydrates, fats and proteins. |
867-81-2 | 20 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Finasteride |
This drug belongs to the 4-nitrogen steroid hormone compound and is a specific type II 5alpha;-reductase competitive inhibitor. It inhibits the conversion of peripheral testosterone into dihydrotestosterone and reduces the levels of dihydrotestosterone in the blood, prostate, skin and other tissues. The growth, development and benign hyperplasia of the prostate depend on dihydrotestosterone. Finasteride inhibits prostate hyperplasia by reducing the levels of dihydrotestosterone in the blood and prostate tissues, and improves the related clinical symptoms of benign prostatic hyperplasia.
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This drug belongs to the 4-nitrogen steroid hormone compound and is a specific type II 5alpha;-reductase competitive inhibitor. It inhibits the conversion of peripheral testosterone into dihydrotestosterone and reduces the levels of dihydrotestosterone in the blood, prostate, skin and other tissues. The growth, development and benign hyperplasia of the prostate depend on dihydrotestosterone. Finasteride inhibits prostate hyperplasia by reducing the levels of dihydrotestosterone in the blood and prostate tissues, and improves the related clinical symptoms of benign prostatic hyperplasia. |
5mg | 98319-26-7 | 51 |