This product is a calcium influx blocker (also known as a calcium channel blocker or calcium ion antagonist), which blocks extracellular calcium ions from entering myocardial and vascular smooth muscle cells through the calcium ion channels (slow channels) of the cell membrane. This product directly relaxes vascular smooth muscle, dilates peripheral arterioles, reduces peripheral resistance (afterload), and dilates coronary arteries, which can relieve coronary artery spasm, and has the effects of anti-hypertension and relieving angina pectoris.

This product is an intermediate in the synthesis of Schisandrae Chinensis A, which can reduce liver damage and alanine aminotransferase (ALT) elevation caused by carbon tetrachloride; inhibit liver microsomal lipid peroxidation caused by carbon tetrachloride, its metabolic conversion to carbon monoxide, and reduce the consumption of reduced coenzyme II and oxygen in the metabolic process, protecting the structure and function of liver cell biomembrane; reduce the increase of liver ALT induced by prednisone, and promote liver regeneration in mice with partial hepatectomy; have a significant induction effect on cytochrome P450 enzyme activity, enhance the detoxification ability of carbon tetrachloride and certain carcinogens; improve protein metabolism in some hepatitis patients, increase albumin and reduce globulin. However, it has no negative conversion effect on HbsAg and HbeAg, and cannot shrink enlarged liver and spleen.

This product is a semi-synthetic 14-membered macrolide antibiotic, and its antibacterial spectrum and antibacterial effect are basically similar to those of erythromycin. It is slightly less effective than erythromycin against Gram-positive bacteria, and stronger than erythromycin against Legionella pneumophila. Its antimicrobial effect against Chlamydia pneumoniae, Mycoplasma pneumoniae, and Ureaplasma urealyticum is similar to or slightly stronger than that of erythromycin. This product can penetrate the bacterial cell membrane and reversibly bind to the 50S subunit of the bacterial ribosome near the donor ("P" position), blocking the transfer RNA (t-RNA) from binding to the "P" position, and also blocking the transfer of the polypeptide chain from the acceptor position ("A" position) to the "P" position, thereby inhibiting bacterial protein synthesis.