Extract from the above information

Extract from the above information

After a healthy person takes 40 mg of this product, it is mainly absorbed through the digestive tract and partially absorbed from the oral mucosa. The blood concentration reaches the peak value in about 2 hours, the peak concentration (Cmax) is about 111.36 ng/ml, and the elimination half-life is about 11.47 hours. This product is metabolized in the liver and excreted from the urine in the form of metabolites.

Iron is a component of hemoglobin in red blood cells. When iron is deficient, the amount of hemoglobin synthesized by red blood cells decreases, causing the red blood cells to become smaller and their oxygen-carrying capacity to decrease, resulting in iron-deficiency anemia. Oral administration of this product can supplement iron and correct iron-deficiency anemia.

It stimulates the adrenergic receptors of the sympathetic nervous system and the dopamine receptors located in the kidney, mesentery, coronary artery, and cerebral artery, and its effect is dose-dependent. At low doses (0.5-2μg/kg per minute based on body weight), it mainly acts on dopamine receptors, dilates the blood vessels of the kidney and mesentery, increases renal blood flow and glomerular filtration rate, and increases urine volume and sodium excretion; at small to medium doses (2-10μg/kg per minute based on body weight), it can directly stimulate β1 receptors and indirectly promote the release of norepinephrine from storage sites, produce positive stress on the myocardium, increase myocardial contractility and stroke volume, and ultimately increase cardiac output, systolic blood pressure, and possibly increase pulse pressure, with no change or slight increase in diastolic blood pressure, and peripheral total resistance often unchanged, and coronary blood flow and oxygen consumption improved; at high doses (more than 10μg/kg per minute based on body weight), it stimulates α receptors, resulting in increased peripheral vascular resistance, renal vasoconstriction, and reduced renal blood flow and urine volume. Due to the increase in cardiac output and peripheral vascular resistance, both systolic and diastolic blood pressures increase. It has a strong effect on stimulating cardiac β1 receptors and increasing myocardial contractility; by increasing the blood flow to the kidneys and mesentery, it can prevent the malignant development of shock caused by ischemia of these organs. Under the same condition of increasing myocardial contractility, the effects of causing arrhythmias and increasing myocardial oxygen consumption are weaker.