Shineway Pharmaceutical Group Ltd.

It has the effects of sedation, anticonvulsant, enhancing hypoxia tolerance, and enhancing the body's immune function.

Honey fungus has similar pharmacological effects to Gastrodia elata. Honey fungus fruiting bodies contain mannitol, lecithin, ergosterol, vitamin A (high content), vitamin B, C and various amino acids. It has pharmacological effects such as sedation, anticonvulsant, enhancing hypoxia tolerance, and enhancing the body's immune function.

It has a strong antibacterial effect on most anaerobic bacteria, and its bactericidal mechanism has not yet been fully elucidated. The nitroreductase of anaerobic bacteria plays an important role in the energy metabolism of sensitive strains. The nitro group of this product is reduced to a cytotoxin, thereby acting on the DNA metabolism process of bacteria and promoting cell death.

It is a long-acting nonsteroidal anti-inflammatory analgesic. After entering the body, it is metabolized into diphenyl ethyl ester, which can inhibit the activity of cyclooxygenase, reduce the synthesis of prostaglandins, and produce anti-inflammatory, analgesic and antipyretic effects.

It has anti-myocardial ischemia effect, can increase coronary blood flow and myocardial nutritional blood flow, and effectively dilate blood vessels; it has lipid-lowering effect, can reduce total cholesterol, glyceryl trinitrate and esterified fatty acids in serum, and has anti-thrombotic and platelet aggregation inhibitory effects; it has brain protective effect, has a protective effect on brain tissue, and has analgesic, sedative, anticonvulsant, anti-inflammatory, uterine excitation, and smooth muscle excitation effects; it can improve the cellular hypoxia state and the internal environment of the body, increase the blood flow to organs such as the heart, brain, and liver, and improve microcirculation.

It has a direct relaxing effect on vascular smooth muscle, reducing external resistance and lowering blood pressure. It has an antispasmodic effect on gastrointestinal smooth muscle.

The mechanism of central nervous system inhibition has not yet been clearly elucidated; it also has an anti-motion sickness effect.

This product is a compound antihypertensive drug. Dibazole has a direct relaxing effect on vascular smooth muscle, which reduces external resistance and lowers blood pressure. It has a spasmolytic effect on gastrointestinal smooth muscle. Reserpine has antihypertensive, slows heart rate and inhibits the central nervous system. In the early stage after administration of hydrochlorothiazide, through diuresis and sodium excretion, the volume of plasma and extracellular fluid is reduced, blood volume and cardiac output are reduced, and thus blood pressure is reduced; when administered for a long time, blood volume and cardiac output can return to their original levels, but total peripheral vascular resistance is reduced and blood pressure can still be reduced. Guanidine sulfate selectively acts on the adrenergic nerve endings after the sympathetic nerves, causing the norepinephrine stored in the nerve endings to be slowly replaced by this product and released. The norepinephrine that should be in the nerve endings and tissues is depleted and missing. This product can also prevent the normal release of norepinephrine during nerve stimulation, weaken the vasoconstriction effect, especially when the body position changes, the sympathetic nerves are sluggish to react, and the excitement that should be reduced, thereby lowering blood pressure. The mechanism of promethazine hydrochloride inhibiting the central nervous system has not been clearly elucidated. In addition, it has an anti-motion sickness effect. The mechanism of action of chlordiazepoxide is related to its selective action on the limbic system of the brain, binding to the central benzodiazepine receptors to promote the release of r-aminobutyric acid and promote synaptic conduction function. This product also has central muscle relaxant and anticonvulsant effects. It has an anti-anxiety effect at low doses. As the dose increases, it can show sedation, hypnosis, and memory impairment. Potassium chloride and calcium lactate in the compound are used to supplement potassium and calcium.

It selectively acts on the postganglionic adrenergic nerve endings of the sympathetic nerves, causing the norepinephrine stored in the nerve endings to be slowly replaced by this product and released. This causes the necessary norepinephrine in the nerve endings and tissues to be depleted and lost. It can also prevent the normal release of norepinephrine during nerve stimulation, weakening the vasoconstriction effect, especially when the body position changes, the sympathetic nerves react slowly, and the necessary excitement is weakened, thereby lowering blood pressure.

This product is a compound antihypertensive drug. Dibazole has a direct relaxing effect on vascular smooth muscle, which reduces external resistance and lowers blood pressure. It has a spasmolytic effect on gastrointestinal smooth muscle. Reserpine has antihypertensive, slows heart rate and inhibits the central nervous system. In the early stage after administration of hydrochlorothiazide, through diuresis and sodium excretion, the volume of plasma and extracellular fluid is reduced, blood volume and cardiac output are reduced, and thus blood pressure is reduced; when administered for a long time, blood volume and cardiac output can return to their original levels, but total peripheral vascular resistance is reduced and blood pressure can still be reduced. Guanidine sulfate selectively acts on the adrenergic nerve endings after the sympathetic nerves, causing the norepinephrine stored in the nerve endings to be slowly replaced by this product and released. The norepinephrine that should be in the nerve endings and tissues is depleted and missing. This product can also prevent the normal release of norepinephrine during nerve stimulation, weaken the vasoconstriction effect, especially when the body position changes, the sympathetic nerves are sluggish to react, and the excitement that should be reduced, thereby lowering blood pressure. The mechanism of promethazine hydrochloride inhibiting the central nervous system has not been clearly elucidated. In addition, it has an anti-motion sickness effect. The mechanism of action of chlordiazepoxide is related to its selective action on the limbic system of the brain, binding to the central benzodiazepine receptors to promote the release of r-aminobutyric acid and promote synaptic conduction function. This product also has central muscle relaxant and anticonvulsant effects. It has an anti-anxiety effect at low doses. As the dose increases, it can show sedation, hypnosis, and memory impairment. Potassium chloride and calcium lactate in the compound are used to supplement potassium and calcium.

Supplement potassium.

Antihypertensive, heart rate slowing and central nervous system depressant effects.

It is related to its selective action on the limbic system of the brain, binding to the central benzodiazepine receptors to promote the release of r-aminobutyric acid and promote synaptic conduction function. It also has central muscle relaxant and anticonvulsant effects, antianxiety effects at low doses, and sedation, hypnosis, and memory impairment as the dose increases.

By excreting sodium through diuresis, the volume of plasma and extracellular fluid is reduced, blood volume and cardiac output are reduced, and thus blood pressure is lowered; when the drug is administered for a long time, total peripheral vascular resistance is reduced and blood pressure can still be lowered.

Supplement calcium.

This product is a compound antihypertensive drug. Dibazole has a direct relaxing effect on vascular smooth muscle, which reduces external resistance and lowers blood pressure. It has a spasmolytic effect on gastrointestinal smooth muscle. Reserpine has antihypertensive, slows heart rate and inhibits the central nervous system. In the early stage after administration of hydrochlorothiazide, through diuresis and sodium excretion, the volume of plasma and extracellular fluid is reduced, blood volume and cardiac output are reduced, and thus blood pressure is reduced; when administered for a long time, blood volume and cardiac output can return to their original levels, but total peripheral vascular resistance is reduced and blood pressure can still be reduced. Guanidine sulfate selectively acts on the adrenergic nerve endings after the sympathetic nerves, causing the norepinephrine stored in the nerve endings to be slowly replaced by this product and released. The norepinephrine that should be in the nerve endings and tissues is depleted and missing. This product can also prevent the normal release of norepinephrine during nerve stimulation, weaken the vasoconstriction effect, especially when the body position changes, the sympathetic nerves are sluggish to react, and the excitement that should be reduced, thereby lowering blood pressure. The mechanism of promethazine hydrochloride inhibiting the central nervous system has not been clearly elucidated. In addition, it has an anti-motion sickness effect. The mechanism of action of chlordiazepoxide is related to its selective action on the limbic system of the brain, binding to the central benzodiazepine receptors to promote the release of r-aminobutyric acid and promote synaptic conduction function. This product also has central muscle relaxant and anticonvulsant effects. It has an anti-anxiety effect at low doses. As the dose increases, it can show sedation, hypnosis, and memory impairment. Potassium chloride and calcium lactate in the compound are used to supplement potassium and calcium.

This product is a compound antihypertensive drug. Dibazole has a direct relaxing effect on vascular smooth muscle, which reduces external resistance and lowers blood pressure. It has a spasmolytic effect on gastrointestinal smooth muscle. Reserpine has antihypertensive, slows heart rate and inhibits the central nervous system. In the early stage after administration of hydrochlorothiazide, through diuresis and sodium excretion, the volume of plasma and extracellular fluid is reduced, blood volume and cardiac output are reduced, and thus blood pressure is reduced; when administered for a long time, blood volume and cardiac output can return to their original levels, but total peripheral vascular resistance is reduced and blood pressure can still be reduced. Guanidine sulfate selectively acts on the adrenergic nerve endings after the sympathetic nerves, causing the norepinephrine stored in the nerve endings to be slowly replaced by this product and released. The norepinephrine that should be in the nerve endings and tissues is depleted and missing. This product can also prevent the normal release of norepinephrine during nerve stimulation, weaken the vasoconstriction effect, especially when the body position changes, the sympathetic nerves are sluggish to react, and the excitement that should be reduced, thereby lowering blood pressure. The mechanism of promethazine hydrochloride inhibiting the central nervous system has not been clearly elucidated. In addition, it has an anti-motion sickness effect. The mechanism of action of chlordiazepoxide is related to its selective action on the limbic system of the brain, binding to the central benzodiazepine receptors to promote the release of r-aminobutyric acid and promote synaptic conduction function. This product also has central muscle relaxant and anticonvulsant effects. It has an anti-anxiety effect at low doses. As the dose increases, it can show sedation, hypnosis, and memory impairment. Potassium chloride and calcium lactate in the compound are used to supplement potassium and calcium.