After binding to nicotinic receptors, this product produces a stable depolarization effect, causing skeletal muscle relaxation. This product can be quickly hydrolyzed by pseudocholinesterase in the blood after entering the body, and its intermediate metabolite succinyl monocholine has a very weak muscle relaxant effect. After intravenous injection, this product first causes a short period of muscle fasciculation, starting from small muscles such as the brow and upper eyelid, to the scapula and pectoralis major, and to the upper and lower limbs. The muscle relaxant effect takes effect in 60 to 90 seconds and lasts for about ten minutes. Repeated intravenous injection or continuous drip can prolong the effect.

After binding to nicotinic receptors, this product produces a stable depolarization effect, causing skeletal muscle relaxation. After intravenous injection, it first causes a short period of muscle fasciculation, starting from small muscles such as the brow and upper eyelid, to the scapula and pectoralis major, and to the upper and lower limbs. The muscle relaxation effect takes effect in 60 to 90 seconds and lasts for about 10 minutes. Large doses can cause a slow heart rate, and arrhythmias such as rhythmic rhythm and premature contractions may also occur. Histamine release may cause bronchospasm or anaphylactic shock. When the dose exceeds 1g, desensitization blockade is likely to occur, delaying the recovery of muscle tone. This product can cause cerebral vasodilation, increased intracranial pressure, orbital smooth muscle contraction, temporary increase in intraocular pressure, postoperative muscle pain, myoglobulinuria, etc.; long-term depolarization can lead to K outflow from muscle cells and increased blood potassium. In addition, this product can induce malignant hyperthermia.

Drowsiness occurs 3 to 5 minutes after intravenous injection, and deep sleep occurs 10 to 15 minutes later. The effect lasts for 90 to 120 minutes, and sometimes can last for several hours. The inhibition of central nervous system activity is mainly due to the excitation of GABA receptors, and has no analgesic effect; it has an excitatory effect on the circulatory system, prompting K to enter cells, which can cause electrocardiogram changes; for the respiratory system, a general dose can slightly slow down the respiratory rate and slightly increase the tidal volume, and a large dose can produce respiratory depression after rapid injection; it can make the throat reflex sluggish, inhibited, and the mandible relaxed, and endotracheal intubation can be performed after surface anesthesia.

A selective beta-2 receptor agonist that has a strong and lasting dilation effect on bronchial smooth muscle

It has certain anti-allergic and bronchial ciliary movement effects

It has an antitussive effect and slightly suppresses the central nervous system.

A selective beta-2 receptor agonist that has a strong and lasting dilation effect on bronchial smooth muscle

It has certain anti-allergic and bronchial ciliary movement effects

It has an antitussive effect and slightly suppresses the central nervous system.