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Zhengzhou Handu Pharmaceutical Group Co., Ltd.
  • Founded in:

    1997-09-27
  • Country:

    China China
  • Address:

    Xincun Town, Xinzheng City, south of Xinmi Railway, east of National Highway 107
  • Tax NO.:

    91410184719125269A
  • Registered Funds:

    50 million yuan
  • Website:

  • Email:

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Qianglong Yishen Tablets
Oyster, dragon bone, Zanthoxylum bungeanum, clove, astragalus, actinolite, deer antler, siler, and cuttlebone.
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Bismuth Potassium Citrate Capsules
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BISMUTH POTASSIUM CITRATE

This product is a gastric mucosal protective agent. Under gastric acid conditions, it forms a diffuse protective layer covering the ulcer surface, preventing gastric acid, enzymes and food from invading the ulcer, and promoting the regeneration of the ulcer mucosa and the healing of the ulcer. This product also has the effects of reducing the activity of pepsin, increasing mucin secretion, and promoting the release of PGE2 from the mucosa, thereby protecting the gastric mucosa. In addition, this product has a killing effect on Helicobacter pylori (Hp), thus promoting the healing of gastritis.

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This product is a gastric mucosal protective agent. Under gastric acid conditions, it forms a diffuse protective layer covering the ulcer surface, preventing gastric acid, enzymes and food from invading the ulcer, and promoting the regeneration of the ulcer mucosa and the healing of the ulcer. This product also has the effects of reducing the activity of pepsin, increasing mucin secretion, and promoting the release of PGE2 from the mucosa, thereby protecting the gastric mucosa. In addition, this product has a killing effect on Helicobacter pylori (Hp), thus promoting the healing of gastritis.

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Gemfibrozil Capsules
Gemfibrozil
Name Description Content CAS NO. Registered Holders
Gemfibrozil

This product is a lipid-regulating drug of the clofibric acid derivative class. Its mechanism of action in lowering blood lipids is not yet fully understood. It may involve peripheral fat decomposition, reduce the liver's uptake of free fatty acids and reduce the formation of triglycerides in the liver, inhibit the synthesis of very low-density lipoprotein apolipoproteins and reduce the production of very low-density lipoproteins. This product reduces blood triglycerides and increases blood high-density lipoprotein concentrations. Although it can slightly reduce blood low-density lipoprotein cholesterol blood concentrations, it may increase low-density lipoprotein in type IV hyperlipoproteinemia. A 5-year placebo-controlled study showed that this product can reduce the occurrence of severe coronary heart disease sudden death and myocardial infarction. Long-term administration of 10 times the human dose to rats increased the incidence of liver malignancies and benign testicular tumors.

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This product is a lipid-regulating drug of the clofibric acid derivative class. Its mechanism of action in lowering blood lipids is not yet fully understood. It may involve peripheral fat decomposition, reduce the liver's uptake of free fatty acids and reduce the formation of triglycerides in the liver, inhibit the synthesis of very low-density lipoprotein apolipoproteins and reduce the production of very low-density lipoproteins. This product reduces blood triglycerides and increases blood high-density lipoprotein concentrations. Although it can slightly reduce blood low-density lipoprotein cholesterol blood concentrations, it may increase low-density lipoprotein in type IV hyperlipoproteinemia. A 5-year placebo-controlled study showed that this product can reduce the occurrence of severe coronary heart disease sudden death and myocardial infarction. Long-term administration of 10 times the human dose to rats increased the incidence of liver malignancies and benign testicular tumors.

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Telmisartan Tablets
The main ingredient of this product is telmisartan.
Name Description Content CAS NO. Registered Holders
Telmisartan

Telmisartan is a specific angiotensin II receptor (ATⅠ type) antagonist, which binds to the ATⅠ receptor subtype with high affinity, has no agonist effect, and has a selective and lasting binding. It does not inhibit human plasma renin, ion channels, and angiotensin converting enzyme Ⅱ, and can almost completely inhibit the increase in blood pressure caused by angiotensin Ⅱ. The antihypertensive effect reaches its maximum 4 weeks after the start of treatment and can be maintained for a long time. Sudden interruption of treatment will not cause rebound hypertension, and the incidence of dry cough is lower than that of angiotensin converting enzyme inhibitors.

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Eliminate rheumatism, relieve pain and dredge meridians

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Nourishes the liver and kidneys, relieves pain and dredges the meridians

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DGL

Regulate bone metabolism and improve body immunity

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Fenugreek

Remove bone spurs, anti-inflammatory and blood circulation

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DIOSCOREAE NIPPONICAE RHIZOMA

Dispel wind and dampness, relieve pain and dredge meridians

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COICIS SEMEN

Eliminate dampness and strengthen the spleen, nourish the liver and kidneys

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