Analgesic, anti-inflammatory, antipyretic, anti-rheumatic, inhibiting platelet aggregation

It has obvious anti-acid effect, and also has gastric mucosal protective effect. It also has a certain adsorption effect on bile acid. Its effect is rapid, mild and long-lasting.

This product has obvious anti-acid effect, and also has gastric mucosal protective effect. It also has a certain adsorption effect on bile acid. Its effect is rapid, mild and long-lasting.

This product has obvious anti-acid effect, and also has gastric mucosal protective effect. It also has a certain adsorption effect on bile acid. Its effect is rapid, mild and long-lasting.

This product has obvious anti-acid effect, and also has gastric mucosal protective effect. It also has a certain adsorption effect on bile acid. Its effect is rapid, mild and long-lasting.

This product has obvious anti-acid effect, and also has gastric mucosal protective effect. It also has a certain adsorption effect on bile acid. Its effect is rapid, mild and long-lasting.

It can increase coronary artery flow and keep blood vessels unobstructed; lower blood pressure and resist arrhythmia; reduce atherosclerosis, reduce intestinal absorption of cholesterol, and prevent fat deposition in tissues; lower cholesterol and triglycerides and promote fat decomposition; comprehensively improve microcirculation and enhance human immunity.

Significantly increase high-density lipoprotein (HDL); comprehensively improve microcirculation and enhance human immunity.

Lowers blood pressure and resists arrhythmia; reduces atherosclerosis, reduces intestinal absorption of cholesterol, and prevents fat deposition in tissues; lowers cholesterol and triglycerides and promotes fat decomposition.

It can increase coronary artery flow and keep blood vessels unobstructed; significantly increase high-density lipoprotein (HDL); comprehensively improve microcirculation and enhance human immunity.

Proton pump inhibitor. This product is a fat-soluble weak alkaline drug that is easily concentrated in an acidic environment. Therefore, after oral administration, it can be specifically distributed in the secretory tubules of the gastric mucosal parietal cells and converted into the active form of sulfenamide in this high-acid environment. It then irreversibly combines with the sulfhydryl group of the H, K-ATPase (also known as the proton pump) in the secretory membrane of the parietal cells through a disulfide bond to form a complex of sulfenamide and the proton pump, thereby inhibiting the activity of the enzyme and blocking the final step of gastric acid secretion. Therefore, this product has a strong and lasting inhibitory effect on gastric acid secretion caused by various reasons.

This product is a long-acting non-steroidal anti-inflammatory drug. After entering the body, it is metabolized into diphenyl ethyl ester, which inhibits the activity of cyclooxygenase and reduces the synthesis of prostaglandins. Animal experiments show that the anti-inflammatory and analgesic effects of this product are weaker than indomethacin, but stronger than aspirin.