No specific description provided

Cephalexin is a first-generation cephalosporin with an antibacterial spectrum similar to that of cephalothin, but its antibacterial activity is poorer than that of the latter. Except for Enterococcus and methicillin-resistant Staphylococci, most strains of Streptococcus pneumoniae, hemolytic Streptococcus, and Staphylococcus aureus that produces or does not produce penicillinase are sensitive to this product. This product has a good antibacterial effect on Neisseria, but Haemophilus influenzae is less sensitive to this product; this product has a certain antibacterial effect on some Escherichia coli, Proteus mirabilis, Salmonella and Shigella. The remaining Enterobacteriaceae, Acinetobacter, Pseudomonas aeruginosa, and Bacteroides fragilis are resistant to this product. Fusobacterium and Veillonella are generally sensitive to this product, and anaerobic Gram-positive cocci are moderately sensitive to this product.

It can directly inhibit the Na-K exchange in the distal tubules and collecting ducts of the kidney, thereby increasing the excretion of Na, Cl, and water, and reducing the excretion of K. Combination with hydrochlorothiazide can enhance the diuretic effect and reduce adverse reactions such as hypokalemia.

Inhibits the reabsorption of sodium chloride in the proximal tubule and distal tubule, increases Na-K exchange in the distal tubule and collecting duct, increases urinary sodium, potassium, chloride, phosphorus and magnesium ion excretion, and reduces urinary calcium excretion; may participate in lowering blood pressure through diuretic natriuretic effect and extrarenal mechanism; affects renal hemodynamics and glomerular filtration function, leading to increased activity of renin-angiotensin system, causing renal vasoconstriction, decreased renal blood flow, and reduced glomerular filtration rate.

This product is a brain metabolism improving drug, which is a cyclic derivative of γ-aminobutyric acid. It has the effect of resisting brain function damage caused by physical and chemical factors. It can promote the conversion of ADP into ATP in the brain, promote the synthesis of acetylcholine and enhance the conduction of nerve excitation, and has the effect of promoting brain metabolism. It can resist brain function damage caused by physical and chemical factors. It has an improving effect on retrograde amnesia caused by hypoxia. It can enhance memory and improve learning ability.

It can significantly inhibit the day and night basal gastric acid secretion, and can also inhibit the gastric acid secretion induced by food, histamine, pentagastrin, caffeine and insulin.