This product is an intermediate in the synthesis of Schisandrae Chinensis A, which can reduce liver damage and alanine aminotransferase (ALT) elevation caused by carbon tetrachloride; it has an inhibitory effect on liver microsomal lipid peroxidation caused by carbon tetrachloride and the metabolic conversion of carbon tetrachloride into carbon monoxide, and reduces the consumption of reduced coenzyme II and oxygen in the metabolic process of carbon tetrachloride, thereby protecting the structure and function of liver cell biomembrane; it can reduce the increase of liver ALT induced by prednisone and promote liver regeneration in mice with partial hepatectomy; it has a significant inducing effect on cytochrome P450 enzyme activity and enhances the detoxification ability of carbon tetrachloride and certain carcinogens; it has an effect on improving protein metabolism in some hepatitis patients, increasing albumin and reducing globulin; it has no negative conversion effect on HbsAg and HbeAg, and cannot shrink enlarged liver and spleen.

Cough and asthma medicine

Cough and asthma medicine

Cough and asthma medicine

Cough and asthma medicine

Cough and asthma medicine

Diclofenac is a non-steroidal anti-inflammatory analgesic derived from phenylacetic acid. Its mechanism of action is to inhibit the activity of cyclooxygenase, thereby blocking the conversion of arachidonic acid into prostaglandins. At the same time, it can also promote the combination of arachidonic acid with triglycerides (triacylglycerols), reduce the concentration of free arachidonic acid in cells, and indirectly inhibit the synthesis of leukotrienes. Diclofenac is a stronger non-steroidal anti-inflammatory drug, and its inhibitory effect on prostaglandin synthesis is stronger than that of aspirin and indomethacin.

Participate in cell energy metabolism and protein synthesis in the body, increase the activity of related metabolic enzymes, improve liver function, and promote the recovery of damaged liver

Inosine participates in cellular energy metabolism and protein synthesis in the body, increases the activity of related metabolic enzymes, improves liver function, and promotes the recovery of damaged liver.

Inosine participates in cellular energy metabolism and protein synthesis in the body, increases the activity of related metabolic enzymes, improves liver function, and promotes the recovery of damaged liver.

Inosine participates in cellular energy metabolism and protein synthesis in the body, increases the activity of related metabolic enzymes, improves liver function, and promotes the recovery of damaged liver.

Sulfonamide antibiotics used for intestinal infections can enhance their antibacterial activity when used in combination with trimethoprim, and convert the antibacterial effect into a bactericidal effect, thereby reducing the production of drug-resistant strains.

When used in combination with sulfonamides, it can double block the bacterial folic acid anabolism, have a synergistic effect, enhance the antibacterial activity of sulfonamides, and convert the antibacterial effect into a bactericidal effect, thereby reducing the production of drug-resistant strains.

Sulfamidine is a sulfonamide antibiotic used for intestinal infections. The combination of trimethoprim and sulfonamide drugs can double block the bacterial folic acid synthesis metabolism, have a synergistic effect, enhance the antibacterial activity of sulfonamide drugs, and convert the antibacterial effect into a bactericidal effect, reducing the generation of drug-resistant strains.

The combined use of trimethoprim and sulfonamides can doubly block the bacterial folate anabolism, have a synergistic effect, enhance the antibacterial activity of sulfonamides, and convert the antibacterial effect into a bactericidal effect, reducing the production of drug-resistant strains.