By inhibiting cyclooxygenase, it selectively inhibits the synthesis of prostaglandins in the hypothalamic temperature regulation center, leading to peripheral vasodilation and sweating to achieve an antipyretic effect; by inhibiting the synthesis and release of prostaglandins, it increases the pain threshold and has an analgesic effect. It is a peripheral analgesic, weaker than aspirin, and is only effective for mild and moderate pain. This product has no obvious anti-inflammatory effect.

By inhibiting cyclooxygenase, it selectively inhibits the synthesis of prostaglandins in the hypothalamic temperature regulation center, leading to peripheral vasodilation and sweating to achieve the effect of antipyretic; by inhibiting the synthesis and release of prostaglandins, it increases the pain threshold and plays an analgesic role. It is a peripheral analgesic, with a stronger effect than acetaminophen, and is only effective for mild and moderate pain. This product has a certain anti-inflammatory effect.

It helps to enhance the antipyretic and analgesic effects, and at the same time has a slight effect of stimulating the central nervous system.

This product is a glucocorticoid drug. It has multiple pharmacological effects such as anti-inflammatory, anti-toxic, anti-shock and immunosuppressive. 1 Anti-inflammatory effect: It has an effect on inflammation caused by various causes except viruses. Glucocorticoids reduce and prevent tissue response to inflammation, thereby reducing the symptoms of inflammation, and can also inhibit the repair of tissues in the late stage of inflammation and reduce sequelae. 2 Immunosuppressive effect: Prevent or inhibit cell-mediated immune responses, delayed allergic reactions, and reduce the expansion of primary immune responses. 3 Anti-toxic and anti-shock effects: Glucocorticoids can improve the body's tolerance, reduce cell damage, and play a role in protecting the body. It also dilates blood vessels, enhances myocardial contractility, and improves microcirculation.

This product is a benzimidazole derivative, which is rapidly metabolized into sulfoxide, sulfone alcohol and 2-amine sulfone alcohol in the body. It selectively and irreversibly inhibits the polymerization of the cytoplasmic microtubule system of the parasite's intestinal wall cells, blocks its absorption of various nutrients and glucose, leads to the depletion of endogenous glycogen in the worm, and inhibits the fumarate reductase system, preventing the production of adenosine triphosphate, making it impossible for the worm to survive and reproduce. It can also cause the degeneration of the cytoplasmic microtubules of the worm's intestinal cells, and bind to its microtubule protein, causing intracellular transport blockage, resulting in the accumulation of Golgi endocrine granules, the gradual dissolution of the cytoplasm, and the complete degeneration of the absorbing cells, causing the death of the worm. It has the effect of completely killing hookworm eggs and whipworm eggs and partially killing ascarid eggs. In addition to killing and expelling various nematodes parasitic in animals, it also has a significant killing and expelling effect on tapeworms and cysticerci.

It can directly stimulate adrenaline receptors, and can also indirectly stimulate adrenaline receptors by prompting adrenaline nerve endings to release norepinephrine. It has a stimulating effect on both alpha and beta receptors. It can dilate bronchi and constrict local blood vessels, and its action time is longer; it strengthens myocardial contractility, increases cardiac output, and makes venous return to the heart sufficient; it has a stronger central nervous system stimulating effect than adrenaline.

It has ① antihistamine effect, which can compete with histamine released from tissues for H1 receptors on effector cells, thereby stopping allergic reactions; ② at the same time, it inhibits central nervous system activity and causes sedative and hypnotic effects; ③ it enhances the effect of antitussive drugs.

This product has good antibacterial effect on superficial fungi such as Trichophyton, Microsporum, Epidermophyton, etc. It has no antibacterial effect on Candida, Cryptococcus, Histoplasma, Sporothrix, Blastomyces, Coccidioides, etc. This drug inhibits the growth of fungi by interfering with the synthesis of fungal nucleic acids.