Inhibits cyclooxygenase activity, thereby blocking the conversion of arachidonic acid to prostaglandins; promotes the combination of arachidonic acid and triglycerides, reduces the concentration of free arachidonic acid in cells, and indirectly inhibits the synthesis of leukotrienes; the inhibitory effect on prostaglandin synthesis is stronger than aspirin and indomethacin, etc.

Antipyretic, analgesic, sedative, anti-inflammatory

It can counteract the allergic reaction of histamine by antagonizing H1 receptors, does not affect histamine metabolism, does not prevent the release of histamine in the body, and has M cholinergic receptor blocking and central inhibition effects

Most of it can be absorbed in the upper part of the small intestine, but enteric-coated tablets are absorbed slowly. Aspirin has a low protein binding rate, and the protein binding rate of hydrolyzed salicylates is 65% to 90%. The t1/2 is 15 to 20 minutes; the length of the t1/2 of salicylates depends on the dose and urine pH. It is about 2 to 3 hours for a small dose, more than 20 hours for a large dose, and 5 to 18 hours for repeated use. Most of it is quickly hydrolyzed into salicylates in the gastrointestinal tract, liver, and blood, and then metabolized in the liver. The metabolites are mainly salicylic acid and glucuronic acid conjugates, and a small part is oxidized to gentisic acid. The blood drug peak is reached 1 to 2 hours after a single dose. The blood drug concentration is 25 to 50 μg/ml for analgesia and antipyretic; 150 to 300 μg/ml for anti-rheumatic and anti-inflammatory. For patients who take large doses of drugs for a long time, because the main metabolic pathway of the drug is saturated, a slight increase in dose can lead to a large change in blood drug concentration. It is excreted from the kidneys as bound metabolites and free salicylic acid. When the dosage is large, the excretion of unmetabolized salicylic acid increases. The pH value of urine affects the excretion rate. In alkaline urine, the excretion rate is accelerated and the amount of free salicylic acid increases, while in acidic urine, the opposite is true.

This product is a brain metabolism improving drug, which is a cyclic derivative of g-aminobutyric acid. It has the effect of resisting brain function damage caused by physical and chemical factors. It can promote ATP in the brain, promote acetylcholine synthesis and positively enhance the conduction of nerve excitement, and has the effect of promoting brain metabolism. It can resist brain function damage caused by physical and chemical factors. It has an improving effect on retrograde amnesia caused by hypoxia. It can enhance memory and improve learning ability.

See the instruction manual for details

Participates in bone formation and reconstruction of bone tissue after fracture, as well as muscle contraction, nerve transmission, coagulation mechanism and reduces capillary permeability, etc.

Participates in bone formation and reconstruction of bone tissue after fracture, as well as muscle contraction, nerve transmission, coagulation mechanism and reduces capillary permeability, etc.