Acetylglutamine is an acetyl compound of glutamine. After passing through the blood-cerebrospinal fluid barrier, it is decomposed into glutamate and γ-aminobutyric acid (GABA). Glutamate is involved in the information transmission of the central nervous system. γ-aminobutyric acid can antagonize the excitability of glutamate, improve nerve cell metabolism, maintain nerve stress resistance, reduce the effect of blood ammonia, and improve brain function.

Acetylglutamine is an acetyl compound of glutamine, which is decomposed into glutamate and gamma-aminobutyric acid (GABA) after passing through the blood-cerebrospinal fluid barrier. Glutamate is involved in the information transmission of the central nervous system. GABA can antagonize the excitability of glutamate, improve nerve cell metabolism, maintain nerve stress ability, reduce the effect of blood ammonia, and improve brain function.

(1) Inhibit plasminogen activator, so that plasminogen cannot be activated into plasmin, thereby inhibiting the dissolution of fibrin and producing a hemostatic effect. (2) Promote the release of active substances by platelets, enhance platelet aggregation and adhesion. Shorten the coagulation time and produce a hemostatic effect. (3) Enhance capillary resistance and reduce capillary permeability, thereby reducing bleeding.

It has a strong affinity for liver cholesterol metabolic enzymes, hinders the inactivation of cortisol and aldosterone, shows obvious corticosteroid-like effects (such as anti-inflammatory, anti-allergic and protective membrane structure, etc.), and has no obvious corticosteroid-like side effects; promotes bile pigment metabolism, reduces the release of ALT and AST; induces γ-IFN and interleukin II, increases NK cell activity and the OKT4/OKT8 ratio and activates the reticuloendothelial system; inhibits the release of histamine from mast cells; inhibits the formation of cell membrane phospholipase A2 (PL-A2) and prostaglandin E2 (PGE2) and granulomatous reactions; inhibits the production and formation of free radicals and lipid peroxides, and reduces the activity of prolyl hydroxylase; regulates calcium ion channels, and protects lysosomal membranes and mitochondria.

Monoammonium glycyrrhizinate has a strong affinity for liver cholesterol metabolism enzymes, thereby hindering the inactivation of cortisol and aldosterone. After use, it shows obvious corticosteroid-like effects, such as anti-inflammatory, anti-allergic and protective membrane structure, etc.; there are no obvious corticosteroid-like side effects. This product can promote bile pigment metabolism, reduce the release of ALT and AST; induce γ-IFN and interleukin II, increase NK cell activity and OKT4/OKT8 ratio and activate the reticuloendothelial system; inhibit mast cell release of histamine; inhibit the formation of cell membrane phospholipase A2 (PL-A2) and prostaglandin E2 (PGE2) and granulomatous reaction; inhibit the production and formation of free radicals and lipid peroxides, reduce the activity of proline hydroxylase; regulate calcium ion channels, protect lysosomal membranes and mitochondria, and reduce cell

Monoammonium glycyrrhizinate has a strong affinity for liver cholesterol metabolism enzymes, thereby hindering the inactivation of cortisol and aldosterone. After use, it shows obvious corticosteroid-like effects, such as anti-inflammatory, anti-allergic and protective membrane structure, etc.; there are no obvious corticosteroid-like side effects. This product can promote bile pigment metabolism, reduce the release of ALT and AST; induce γ-IFN and interleukin II, increase NK cell activity and OKT4/OKT8 ratio and activate the reticuloendothelial system; inhibit mast cell release of histamine; inhibit the formation of cell membrane phospholipase A2 (PL-A2) and prostaglandin E2 (PGE2) and granulomatous reaction; inhibit the production and formation of free radicals and lipid peroxides, reduce the activity of proline hydroxylase; regulate calcium ion channels, protect lysosomal membranes and mitochondria, and reduce cell

Monoammonium glycyrrhizinate has a strong affinity for liver cholesterol metabolism enzymes, thereby hindering the inactivation of cortisol and aldosterone. After use, it shows obvious corticosteroid-like effects, such as anti-inflammatory, anti-allergic and protective membrane structure, etc.; there are no obvious corticosteroid-like side effects. This product can promote bile pigment metabolism, reduce the release of ALT and AST; induce γ-IFN and interleukin II, increase NK cell activity and OKT4/OKT8 ratio and activate the reticuloendothelial system; inhibit mast cell release of histamine; inhibit the formation of cell membrane phospholipase A2 (PL-A2) and prostaglandin E2 (PGE2) and granulomatous reaction; inhibit the production and formation of free radicals and lipid peroxides, reduce the activity of proline hydroxylase; regulate calcium ion channels, protect lysosomal membranes and mitochondria, and reduce cell

This product is a commonly used contrast agent for X-ray and CT examinations, and can be used in blood vessels, spinal canals, and body cavities. Animal test results show that this product has an enhancement effect on dog liver, abdominal aorta, and CT scan images.