This product is a second-generation sulfonylurea antidiabetic drug, which is effective for most patients with type 2 diabetes. It can reduce fasting and postprandial blood sugar and reduce glycosylated hemoglobin (HbAlc) by 1% to 2%. The main function of this type of drug is to stimulate pancreatic b cells to secrete insulin, but the prerequisite is that pancreatic b cells still have a certain function of synthesizing and secreting insulin. Its mechanism is to specifically bind to the sulfonylurea receptor on the b cell membrane, thereby closing the K channel, causing changes in membrane potential, opening the Ca2 channel, and increasing intracellular Ca2, which promotes insulin secretion. In addition, there are extra-pancreatic effects, including improving the insulin resistance of peripheral tissues (such as liver, muscle, and fat).

This product is a semi-synthetic ergot alkaloid derivative. It has alpha-receptor blocking and vasodilation effects. It can enhance the metabolism of brain cell energy and increase the utilization of oxygen and glucose. It promotes the conversion of neurotransmitter dopamine to increase nerve conduction, strengthens the synthesis of brain protein, and improves brain function.

Because the thiol group in its chemical structure can break the disulfide bonds of mucin, it can reduce sputum viscosity and make sputum easier to cough up.

It has a strong mucolytic effect by decomposing mucin complexes and nucleic acids, changing the purulent components in sputum and other mucus and mucus secretions from viscous to thin; it has a direct antioxidant effect through free sulfhydryl groups (-SH); it protects α-1 antitrypsin from being inactivated by hypochlorous acid; it forms L-cysteine and synthesizes glutathione (GSH) in cells to prevent cells from being damaged by oxygen free radicals and cytotoxic substances; it helps protect cells from cytotoxic damage caused by low GSH levels in the body; it can be used as a special antidote in acetaminophen poisoning, cyclophosphamide treatment and hemorrhagic cystitis.

Nonsteroidal anti-inflammatory drugs (NSAIDs) can inhibit the synthesis of prostaglandins and have analgesic, anti-inflammatory and antipyretic effects. They are absorbed faster than ibuprofen and can achieve analgesic effects 15 to 30 minutes after taking the drug. They can also play analgesic and anti-inflammatory roles and have good therapeutic effects.