Zinc participates in the synthesis and activation of many enzymes, and plays an important physiological role in the synthesis of proteins and nucleic acids, and the absorption and digestion of intestinal proteins. Zinc can promote growth and development, maintain normal appetite and taste by synthesizing taste buds and preventing incomplete keratinization of buccal mucosal epithelial cells, and enhance the level of free radicals in phagocytes. Free radicals can destroy the cell membrane of microorganisms, play a bactericidal role, and accelerate the healing of wounds, burns, and ulcers; Zinc plays an important role in the metabolism of vitamin A and vision, promotes and maintains sexual function, stabilizes cell membranes, improves tissue energy metabolism and tissue respiration, and zinc ions can precipitate proteins.

The vasodilation effect is similar to that of nitroglycerin, but the effect is faster. It releases nitric oxide (NO), which is the same as endothelial relaxing factor, activates guanylate cyclase, increases cyclic guanosine monophosphate (cGMP) in smooth muscle and other tissues, and leads to vasodilation; it can also dilate peripheral veins, store blood in peripheral veins, reduce left ventricular end pressure and reduce coronary blood flow resistance during diastole, and dilate peripheral arterioles to reduce peripheral resistance and blood pressure, thereby reducing myocardial oxygen consumption and relieving angina pectoris. It also has the effect of relieving cyanide toxicity. Its mechanism is the same as that of sodium nitrite, oxidizing divalent iron (Fe2) in hemoglobin to trivalent iron (Fe3), and Fe3 then combines with cyanide (CN-) to form methemoglobin, temporarily delaying the toxicity of cyanide. It takes effect 30 seconds after inhalation and lasts for 3 to 5 minutes. After taking the drug, the heart rate increases, blood pressure decreases, and left ventricular end-diastolic pressure decreases.

It releases nitric oxide (NO), activates guanylate cyclase, increases cyclic guanosine monophosphate (cGMP) in smooth muscle and other tissues, and leads to vasodilation. It can also dilate peripheral veins, reduce left ventricular end pressure and diastolic coronary blood flow resistance, and dilate arterioles to lower blood pressure, thereby reducing myocardial oxygen consumption and relieving angina pectoris. It also has the effect of relieving cyanide toxicity, delaying toxicity by oxidizing divalent iron into trivalent iron and combining it with cyanide to form methemoglobin.

It releases nitric oxide (NO), activates guanylate cyclase, increases cyclic guanosine monophosphate (cGMP) in smooth muscle and other tissues, and leads to vasodilation. It can also dilate peripheral veins, reduce left ventricular end pressure and diastolic coronary blood flow resistance, and dilate arterioles to lower blood pressure, thereby reducing myocardial oxygen consumption and relieving angina pectoris. It also has the effect of relieving cyanide toxicity, delaying toxicity by oxidizing divalent iron into trivalent iron and combining it with cyanide to form methemoglobin.

The vasodilation effect is similar to that of nitroglycerin, but the effect is faster. It releases nitric oxide (NO), which is the same as endothelial relaxing factor, activates guanylate cyclase, increases cyclic guanosine monophosphate (cGMP) in smooth muscle and other tissues, and leads to vasodilation; it can also dilate peripheral veins, store blood in peripheral veins, reduce left ventricular end pressure and reduce coronary blood flow resistance during diastole, and dilate peripheral arterioles to reduce peripheral resistance and blood pressure, thereby reducing myocardial oxygen consumption and relieving angina pectoris. It also has the effect of relieving cyanide toxicity. Its mechanism is the same as that of sodium nitrite, oxidizing divalent iron (Fe2) in hemoglobin to trivalent iron (Fe3), and Fe3 then combines with cyanide (CN-) to form methemoglobin, temporarily delaying the toxicity of cyanide. Sodium nitrite and sodium thiosulfate need to be injected immediately. It takes effect 30 seconds after inhalation and lasts for 3 to 5 minutes. After taking the medicine, the heart rate increases, blood pressure decreases, and left ventricular end-diastolic pressure decreases.

No specific pharmacological effects mentioned

Relaxes vascular smooth muscle, activates guanylate cyclase by releasing nitric oxide (NO), increases cyclic guanosine monophosphate (cGMP), leads to dephosphorylation of myosin light chain, regulates the contraction state of smooth muscle, causes vasodilation, reduces preload and afterload, reduces myocardial oxygen consumption, relieves angina, and dilates epicardial coronary artery branches.