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I. Muscle Synthesis and Muscle Cell Repair (In vitro Skeletal Muscle Model)
Strongly promote myosin synthesis
Upregulate the mTOR pathway, accelerate amino acid absorption and protein assembly in muscle cells, and increase myofiber protein deposition; at the same time, inhibit muscle protein breakdown and reduce muscle loss, used for in vitro studies on muscle atrophy and aging sarcopenia.
Activate muscle stem cell proliferation
Stimulate satellite cell differentiation to produce new muscle cells, repair damaged muscle fibers; in models of exercise injury and aging muscle cells, shorten the cell repair cycle and alleviate muscle cell fatigue and necrosis.
Maintain muscle cell energy homeostasis
Increase muscle mitochondrial generation and oxidative energy efficiency, reduce lactate accumulation, and improve indicators related to muscle cell endurance.
II. Bone and Cartilage Tissue Maintenance (In vitro Bone / Cartilage Cells)
Promote osteoblast activity
Stimulate osteoblast proliferation and collagen synthesis in bone matrix, enhance bone mineralization ability; inhibit excessive activity of osteoclasts, reduce bone loss, commonly used tool in studies of osteoporosis mechanism.
Cartilage matrix repair
Increase secretion of type II collagen and glycosaminoglycans by chondrocytes, repair worn cartilage tissue; reduce apoptosis of chondrocytes induced by inflammation, used in basic experiments on joint degeneration.
III. Bidirectional Regulation of Glycolipid Metabolism (Fat, Liver Cell In Vitro Experiment)
Enhance cellular insulin sensitivity
Promote expression of glucose transport proteins in cell membranes, accelerate sugar uptake and glycogen storage without insulin dependence, improve insulin resistance cell state induced by high sugar and high fat.
Regulate fat metabolism balance
Moderately promote lipolysis and breakdown of triglycerides in adipocytes; at low concentrations, do not excessively consume muscles, distinguish as lipolytic substances; reduce accumulation of triglycerides in cells.
Regulate lipid-related cell pathways
In liver cell models, assist in downregulating low-density lipoprotein deposition signals, used for in vitro mechanism studies of metabolic syndrome.
IV. Broad-spectrum Anti-apoptosis and Cell Survival Protection (Various Body Cells)
Block endogenous cell apoptosis pathways
Stabilize mitochondrial membrane potential, inhibit the release of pro-apoptotic factors, significantly reduce programmed cell death caused by oxidative stress, hypoxia, toxins, and inflammation.
Broad-spectrum cell repair
Beneficial to various body cells such as cardiac, neural, liver, kidney, and skin cells, reduce cell necrosis caused by various stresses, mostly used for screening ischemia and organ injury models.
V. Neurocell Protection and Cognitive-related In Vitro Effects
Neuron nutrition support
As a neurotrophic peptide, promote neuron survival and synaptic growth; reduce neuronal loss caused by oxidative stress in the brain.
Improve the vitality of aging neurons
Enhance mitochondrial function of brain cells, regulate neurotransmitter secretion, used for mechanism studies of brain aging and mild neurodegenerative diseases.
Assist in nerve injury repair
Promote expression of signals for nerve cell migration and regeneration in isolated nerve tissue.
VI. Skin Cell Regeneration and Barrier Repair (Epidermis, Fibroblasts)
Stimulate collagen and elastic fiber synthesis
Activate dermal fibroblasts, increase type I/III collagen production, maintain the integrity of extracellular matrix, reduce collagen degradation caused by oxidation.
Accelerate epidermal cell renewal and repair
Promote proliferation and migration of keratinocytes, accelerate the healing speed of isolated skin wounds; resist damage to epidermal cells caused by ultraviolet free radicals.
Stabilize the skin cell antioxidant system
Upregulate Nrf2 antioxidant pathways, reduce chronic low-grade inflammation in the skin, alleviate cell damage caused by photoaging.
VII. Cardiac, Liver, Kidney Cell Protection (In vitro Organ Sections)
Cardiovascular endothelial protection
Repair vascular endothelial cells, reduce oxidative inflammatory damage, inhibit signals related to abnormal lipid deposition in blood vessels; in ischemic myocardial models, reduce the proportion of myocardial cell apoptosis.
Liver and kidney anti-injury effects
Relieve damage to liver cells and renal tubular cells caused by chemical toxins and oxidative stress, reduce activation of organ fibrosis pathways, used for drug organ toxicity detection experiments.
Hazard Identification
Classification of the substance or mixture
GHS label elements, including precautionary statements
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Other hazards which do not result in classification
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Handling and Storage
Precautions for safe handling
Handling in a well ventilated place. Wear suitable protective clothing. Avoid contact with skin and eyes. Avoid formation of dust and aerosols. Use non-sparking tools. Prevent fire caused by electrostatic discharge steam.
Conditions for safe storage, including any incompatibilities
Store the container tightly closed in a dry, cool and well-ventilated place. Store apart from foodstuff containers or incompatible materials.
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