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Home > Drugs > Honz Pharmaceutical Co., Ltd.
Honz Pharmaceutical Co., Ltd.
  • Founded in:

    1994-01-20
  • Country:

    China China
  • Address:

    No. 6, Yaogu 3rd Road, Yaogu Industrial Park, Haikou National High-tech Industrial Development Zone, Hainan Province
  • Tax NO.:

    91460000708873536T
  • Registered Funds:

    455.124411 yuan
  • Website:

  • Email:

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CITRI GRANDIS EXOCARPIUM

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Citri reticulatae pericarpium

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aster

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Ophiopogonis Radix

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Bitter almonds (peeled and fried)

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Perilla seeds (fried)

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Dextrin

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Levocetirizine Hydrochloride Granules
Levocetirizine hydrochloride. Chemical name: R-(-)-2-[2-[4-[(4-chlorophenyl)benzyl]-1-piperazinyl]ethoxy]acetic acid dihydrochloride Molecular weight: C21H25ClN2O3bull;2HCl
Name Description Content CAS NO. Registered Holders
Levocetirizine dihydrochloride

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Cold and Heat Clearing Granules
Nepeta spicate, mint, siler, bupleurum, perilla leaf, kudzu root, platycodon, bitter almond, angelica dahurica, bitter herb, reed root. Sucrose and dextrin are auxiliary materials.
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SCHIZONEPETAE SPICA

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PEPPERMINT

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SAPOSHNIKOVIAE RADIX

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Bupleurum

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Folia perillae acutae

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Puerariae Lobatae Radix

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Platycodonis Radix

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ARMENIACAE SEMEN AMARUM

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Angelicae Dahuricae Radix

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CORYDALIS BUNGEANAE HERBA

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PHRAGMITIS RHIZOMA

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Sucrose

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Dextrin

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Lactobacillus Granules
1g according to lactobacillus
Name Description Content CAS NO. Registered Holders
Lactobacillin

It forms a protective layer in the intestine to prevent the invasion of pathogens and viruses; stimulates the intestine to secrete antibodies and improves intestinal immunity; selectively kills intestinal pathogens, protects and promotes the growth of beneficial bacteria; regulates the electrolyte and water balance of the intestinal mucosa; promotes the secretion of gastric juice and enhances digestive function.

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It forms a protective layer in the intestine to prevent the invasion of pathogens and viruses; stimulates the intestine to secrete antibodies and improves intestinal immunity; selectively kills intestinal pathogens, protects and promotes the growth of beneficial bacteria; regulates the electrolyte and water balance of the intestinal mucosa; promotes the secretion of gastric juice and enhances digestive function.

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Omeprazole Sodium for Injection
Omeprazole sodium.
Name Description Content CAS NO. Registered Holders
Omeprazole sodium

This product is a proton pump inhibitor of gastric parietal cells. It can specifically inhibit the secretory microtubules and tubular vesicles in the cytoplasm of the parietal cell apical membrane, thereby effectively inhibiting the secretion of gastric acid. Since H, K-ATPase is the last process of acid secretion of parietal cells, this product has a strong acid inhibition ability. It can not only non-competitively inhibit gastric acid secretion caused by gastrin, histamine, choline, food, and stimulation of the vagus nerve, but also inhibit part of the basic gastric acid secretion that is not affected by choline or H2 receptor blockers. It also has a strong and lasting inhibitory effect on gastric acid secretion caused by dibutyl cyclic adenosine monophosphate (DCAMP) stimulation that cannot be inhibited by H2 receptor antagonists. In addition, this product also has an inhibitory effect on pepsin secretion, has no obvious change in gastric mucosal blood flow, and does not affect body temperature, gastric cavity temperature, arterial blood pressure, venous hemoglobin, arterial oxygen partial pressure, carbon dioxide partial pressure and arterial blood pH.

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This product is a proton pump inhibitor of gastric parietal cells. It can specifically inhibit the secretory microtubules and tubular vesicles in the cytoplasm of the parietal cell apical membrane, thereby effectively inhibiting the secretion of gastric acid. Since H, K-ATPase is the last process of acid secretion of parietal cells, this product has a strong acid inhibition ability. It can not only non-competitively inhibit gastric acid secretion caused by gastrin, histamine, choline, food, and stimulation of the vagus nerve, but also inhibit part of the basic gastric acid secretion that is not affected by choline or H2 receptor blockers. It also has a strong and lasting inhibitory effect on gastric acid secretion caused by dibutyl cyclic adenosine monophosphate (DCAMP) stimulation that cannot be inhibited by H2 receptor antagonists. In addition, this product also has an inhibitory effect on pepsin secretion, has no obvious change in gastric mucosal blood flow, and does not affect body temperature, gastric cavity temperature, arterial blood pressure, venous hemoglobin, arterial oxygen partial pressure, carbon dioxide partial pressure and arterial blood pH.

95510-70-6 25
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