This product is a penicillin antibiotic, which has good antibacterial activity against Streptococcus such as Streptococcus pneumoniae and hemolytic Streptococcus, aerobic Gram-positive cocci such as non-penicillinase-producing Staphylococcus and Enterococcus faecalis, aerobic Gram-negative bacteria such as Escherichia coli, Proteus mirabilis, Salmonella, Haemophilus influenzae, Neisseria gonorrhoeae, and Helicobacter pylori. Amoxicillin exerts its bactericidal effect by inhibiting the synthesis of bacterial cell walls, which can quickly dissolve and rupture bacteria into spherical bodies.

This product belongs to the macrolide antibiotics. It inhibits bacterial protein synthesis by binding to the 50S subunit of the ribosome of sensitive bacteria. It has antibacterial activity against Staphylococcus (except methicillin-resistant strains), all groups of streptococci and Gram-positive bacilli. Streptococcus pyogenes, group A hemolytic streptococci, Staphylococcus, Streptococcus pneumoniae, meningococci, gonorrhea, Bordetella pertussis, etc. are also sensitive to this product. This product also has antibacterial effects on various anaerobic bacteria except Bacteroides fragilis and Fusobacterium. It also has inhibitory effects on Mycoplasma pneumoniae, Chlamydia, Legionella, Treponema pallidum, Leptospira, and Campylobacter fetus.

This product is a neutral compound of aspirin and acetaminophen bonded by an ester bond. It has antipyretic and analgesic effects, with fewer adverse reactions than aspirin, and is easily tolerated by patients. After oral administration, it is not hydrolyzed in the gastrointestinal tract, is absorbed in the intestine and quickly reaches an effective concentration in the blood, and is characterized by rarely causing gastrointestinal bleeding.

Overdose can cause hypotension, which should be stopped immediately and volume expansion should be used to correct it. Hemodialysis can also be used to remove it in adults.

It competes with p-aminobenzoic acid for dihydrofolate synthase, inhibiting bacterial dihydrofolate synthesis; combined with trimethoprim, it can double block the bacterial tetrahydrofolate synthesis process and enhance the antibacterial effect.

It competes with p-aminobenzoic acid for dihydrofolate synthase, inhibiting bacterial dihydrofolate synthesis; combined with trimethoprim, it can double block the bacterial tetrahydrofolate synthesis process and enhance the antibacterial effect.

By inhibiting the bacterial dihydrofolate reductase, it hinders the reduction of dihydrofolate into tetrahydrofolate; when used in combination with sulfonamides, it can double block the bacterial tetrahydrofolate synthesis process and enhance the antibacterial effect.