Expand blood volume, improve microcirculation, and prevent thrombosis

Acts as a solvent and assists in maintaining osmotic pressure, indirectly supporting drug efficacy

It has anti-thrombotic effect. It inhibits platelet aggregation and release; inhibits adenosine uptake, increases cyclic adenosine monophosphate (cAMP) in platelets; inhibits phosphodiesterase (PDE), enhances the increase in cGMP concentration caused by endothelial relaxing factor (EDRF); inhibits the formation of thromboxane A2 (TXA2); enhances the effect of endogenous PGI2; and has a dilating effect on blood vessels.

Diclofenac is a non-steroidal anti-inflammatory analgesic derived from phenylacetic acid. Its mechanism of action is to inhibit the activity of cyclooxygenase, thereby blocking the conversion of arachidonic acid into prostaglandins. At the same time, it can also promote the combination of arachidonic acid with triglycerides (triacylglycerols), reduce the concentration of free arachidonic acid in cells, and indirectly inhibit the synthesis of leukotrienes. Diclofenac is a stronger non-steroidal anti-inflammatory drug, and its inhibitory effect on prostaglandin synthesis is stronger than that of aspirin and indomethacin.

An important component of coenzymes required for sugar metabolism

Important coenzyme component required for tissue respiration

A cofactor for many enzymes, involved in the metabolism of amino acids and fats

Component of coenzymes I and II, essential for lipid metabolism and oxidation of tissue respiration

Component of coenzyme A, involved in the metabolism of sugar, fat and protein

It is a non-steroidal anti-inflammatory drug with analgesic, anti-inflammatory and antipyretic effects. This product exerts its pharmacological effects by inhibiting cyclooxygenase, reducing the synthesis of prostaglandins in local tissues, inhibiting the chemotaxis of leukocytes and the release of lysosomal enzymes.