Reduce liver damage and alanine aminotransferase (ALT) elevation caused by carbon tetrafluoride; inhibit liver microsomal lipid peroxidation caused by carbon tetrachloride, carbon tetrachloride metabolism into carbon monoxide, and reduce the consumption of reduced coenzyme II and oxygen in the process of carbon tetrachloride metabolism, thereby protecting the structure and function of liver cell biomembrane; reduce prednisone-induced liver ALT elevation; promote liver regeneration in mice with partial hepatectomy; have a significant induction effect on cytochrome P450 enzyme activity, thereby enhancing the detoxification ability of carbon tetrachloride and certain carcinogens; improve protein metabolism in some hepatitis patients, increase albumin and reduce globulin. It has no negative conversion effect on HbsAg and HbeAg, and cannot shrink enlarged liver and spleen.

Phosphorus supplements are involved in bone formation, cell membrane composition and energy metabolism, and are necessary for cell tissue metabolism in the body.

Acts on the cerebral cortex, promotes mental excitement, relieves fatigue, accelerates cerebral blood circulation, and improves brain function

This product can block the uncoating of influenza A virus and the release of its nucleic acid into respiratory epithelial cells, and also affect the initial replication of the virus that has already penetrated the cells. Since this product works between the virus and the cell and has no host specificity, it is more effective for prevention or early exposure to the virus.

Broad-spectrum antiviral drug. It has the effect of inhibiting the growth of multiple viruses such as respiratory syncytial virus, influenza virus, hepatitis A virus, adenovirus, etc. in vitro. After entering the virus-infected cells, the drug is rapidly phosphorylated, and its product acts as a competitive inhibitor of viral synthase, inhibiting inosine monophosphate dehydrogenase, influenza virus RNA polymerase and mRNA guanosine transferase, thereby causing a decrease in intracellular guanosine triphosphate, impairing viral RNA and protein synthesis, and inhibiting viral replication and transmission. It may also have an immune effect and neutralizing antibody effect on respiratory syncytial virus.

This product can block the uncoating of influenza A virus and the release of its nucleic acid into respiratory epithelial cells, and also affect the initial replication of the virus that has already penetrated into the cells. Since this product works between the virus and the cell and has no host specificity, it is more effective for prevention or early use of the drug in contact with the virus.