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This product is a semi-synthetic aminoglycoside antibiotic aqueous solution for external use, which is stable to the aminoglycoside inactivating enzyme produced by drug-resistant strains. It is used for infectious diseases caused by drug-resistant Pseudomonas aeruginosa, Proteus and Escherichia coli. It can cut off the destructive effect of the aminoacylase of drug-resistant bacteria on drugs, and has a high efficiency against clinical drug-resistant bacteria. When sprayed on the wound, it can prevent bacterial infection and has no irritation to the wound. In an infected wound, when the bacteria come into contact with the drug, they are quickly killed, which accelerates the healing of the wound. This product is effective for infections caused by various sensitive bacteria.

Sodium copper chlorophyllin is a hematopoietic cell complex that may promote the release of neutrophils by increasing the excitability of sympathetic nerves and combining liver and adrenal functions; because its porphyrin ring structure is similar to hemoglobin, it can promote hematopoiesis. Animal experiments have shown that it can improve multiple blood indicators of mice with aplastic anemia, and promote the recovery and regulation of multipotent stem cells, granulocyte-monocyte progenitor cells, erythroid progenitor cells and bone marrow stromal cells. In addition, clinical studies have shown that this product can activate liver reticuloendothelial cells, enhance liver cell resistance, accelerate repair and regeneration, and shrink or eliminate enlarged liver.

This product is a formyl derivative of tetrahydrofolate, mainly used for the rescue of high-dose methotrexate and other folic acid antagonists. The main function of methotrexate is to bind to dihydrofolate reductase, blocking the conversion of dihydrofolate into tetrahydrofolate, thereby inhibiting DNA synthesis. After entering the body, this product is converted into tetrahydrofolate by tetrahydrofolate reductase, which can effectively counteract the toxic reaction caused by methotrexate, but has no obvious effect on the existing methotrexate neurotoxicity.

It increases the body weight of rats with streptozotocin-induced diabetes model, reduces HbA1c and blood glucose levels, reduces whole blood viscosity, plasma viscosity, whole blood reduced viscosity, red blood cell rigidity index, red blood cell aggregation index and fibrinogen content; alleviates the degree of retinal vascular lesions; reduces the contents of glucose, sorbitol and fructose in retinal tissue; increases the amplitude of a wave and b wave of flash electroretinogram, shortens the peak latency of b wave, increases the amplitude of retinal oscillation potential and shortens the peak latency.

It increases the body weight of rats with streptozotocin-induced diabetes model, reduces HbA1c and blood glucose levels, reduces whole blood viscosity, plasma viscosity, whole blood reduced viscosity, red blood cell rigidity index, red blood cell aggregation index and fibrinogen content; alleviates the degree of retinal vascular lesions; reduces the contents of glucose, sorbitol and fructose in retinal tissue; increases the amplitude of a wave and b wave of flash electroretinogram, shortens the peak latency of b wave, increases the amplitude of retinal oscillation potential and shortens the peak latency.

It increases the body weight of rats with streptozotocin-induced diabetes model, reduces HbA1c and blood glucose levels, reduces whole blood viscosity, plasma viscosity, whole blood reduced viscosity, red blood cell rigidity index, red blood cell aggregation index and fibrinogen content; alleviates the degree of retinal vascular lesions; reduces the contents of glucose, sorbitol and fructose in retinal tissue; increases the amplitude of a wave and b wave of flash electroretinogram, shortens the peak latency of b wave, increases the amplitude of retinal oscillation potential and shortens the peak latency.

It increases the body weight of rats with streptozotocin-induced diabetes model, reduces HbA1c and blood glucose levels, reduces whole blood viscosity, plasma viscosity, whole blood reduced viscosity, red blood cell rigidity index, red blood cell aggregation index and fibrinogen content; alleviates the degree of retinal vascular lesions; reduces the contents of glucose, sorbitol and fructose in retinal tissue; increases the amplitude of a wave and b wave of flash electroretinogram, shortens the peak latency of b wave, increases the amplitude of retinal oscillation potential and shortens the peak latency.

It increases the body weight of rats with streptozotocin-induced diabetes model, reduces HbA1c and blood glucose levels, reduces whole blood viscosity, plasma viscosity, whole blood reduced viscosity, red blood cell rigidity index, red blood cell aggregation index and fibrinogen content; alleviates the degree of retinal vascular lesions; reduces the contents of glucose, sorbitol and fructose in retinal tissue; increases the amplitude of a wave and b wave of flash electroretinogram, shortens the peak latency of b wave, increases the amplitude of retinal oscillation potential and shortens the peak latency.

It increases the body weight of rats with streptozotocin-induced diabetes model, reduces HbA1c and blood glucose levels, reduces whole blood viscosity, plasma viscosity, whole blood reduced viscosity, red blood cell rigidity index, red blood cell aggregation index and fibrinogen content; alleviates the degree of retinal vascular lesions; reduces the contents of glucose, sorbitol and fructose in retinal tissue; increases the amplitude of a wave and b wave of flash electroretinogram, shortens the peak latency of b wave, increases the amplitude of retinal oscillation potential and shortens the peak latency.

It increases the body weight of rats with streptozotocin-induced diabetes model, reduces HbA1c and blood glucose levels, reduces whole blood viscosity, plasma viscosity, whole blood reduced viscosity, red blood cell rigidity index, red blood cell aggregation index and fibrinogen content; alleviates the degree of retinal vascular lesions; reduces the contents of glucose, sorbitol and fructose in retinal tissue; increases the amplitude of a wave and b wave of flash electroretinogram, shortens the peak latency of b wave, increases the amplitude of retinal oscillation potential and shortens the peak latency.

It increases the body weight of rats with streptozotocin-induced diabetes model, reduces HbA1c and blood glucose levels, reduces whole blood viscosity, plasma viscosity, whole blood reduced viscosity, red blood cell rigidity index, red blood cell aggregation index and fibrinogen content; alleviates the degree of retinal vascular lesions; reduces the contents of glucose, sorbitol and fructose in retinal tissue; increases the amplitude of a wave and b wave of flash electroretinogram, shortens the peak latency of b wave, increases the amplitude of retinal oscillation potential and shortens the peak latency.