It is hydrolyzed by phosphatase or phosphatase in the liver or tumor in the body and becomes activated phosphoramide nitrogen mustard. Its mechanism of action is to cross-link with DNA, inhibit DNA synthesis, and interfere with RNA function. It is a non-specific drug for the cell cycle. It has a broad anti-tumor spectrum and has inhibitory effects on many tumors.

It relaxes vascular smooth muscle, activates guanylate cyclase by releasing nitric oxide (NO), and increases cyclic guanosine monophosphate (cGMP) in smooth muscle cells, thereby dilating peripheral arteries and veins, reducing the amount of blood returning to the heart, lowering left ventricular end-diastolic pressure, pulmonary capillary wedge pressure (preload), peripheral vascular resistance, systolic arterial pressure and mean arterial pressure (afterload), while dilating coronary arteries to increase coronary perfusion and reduce myocardial oxygen consumption.

Immune bidirectional regulatory drug. It has the function of inducing and promoting the differentiation, maturation and activation of T lymphocytes and their subgroups, regulating the ratio of T lymphocytes to make CD4/CD8 tend to normal; regulating and enhancing the cellular immune function of the human body, which can promote the maturation of T lymphocytes in peripheral blood after mitogen activation, increase the secretion of various lymphokines (such as alpha, gamma interferon, interleukin 2 or interleukin 3) after T cells are activated by various antigens or mitogens, and increase the level of lymphokine receptors on T cells. It also enhances lymphocyte response by activating T helper cells. In addition, this product may affect the chemotaxis of NK precursor cells, which become more cytotoxic after exposure to interferon, which is conducive to its therapeutic effect. In addition, this product can enhance the body's ability to resist radiation.

It is an azole antifungal drug, which has antibacterial and bactericidal effects on dermatophytes, yeasts (Candida, Pityrosporum, Torulopsis, Cryptococcus), dimorphic fungi and Fungi; except for Entomophthora, it has weaker effects on Aspergillus, Schenckia, some Phaeochromocytaceae and Mucor. The mechanism of action of ketoconazole is mainly to highly selectively interfere with the activity of fungal cytochrome P-450, thereby inhibiting the biosynthesis of ergosterol on the fungal cell membrane.

It is a powerful external corticosteroid with rapid effects, strong capillary contraction and anti-inflammatory effects.

Ketoconazole is an azole antifungal drug. It has antibacterial and bactericidal effects on dermatophytes, yeasts (Candida, Pityrosporum, Torulopsis, Cryptococcus), dimorphic fungi and Fungi; except for Entomophthora, it has weaker effects on Aspergillus, Schenckia, some Phaeochromocytaceae and Mucor. The mechanism of action of ketoconazole is mainly to highly selectively interfere with the activity of fungal cytochrome P-450, thereby inhibiting the biosynthesis of ergosterol on the fungal cell membrane. Clobetasol propionate is a potent topical corticosteroid. It acts quickly and has strong capillary constriction and anti-inflammatory effects.

A new type of fluoroquinolone antibiotic that achieves its antibacterial effect by inhibiting the activity of bacterial DNA gyrase and topoisomerase IV, thereby hindering bacterial DNA replication; it is effective against aerobic bacteria, anaerobic bacteria, Gram-positive bacteria and Gram-negative bacteria; it has a killing effect on Pseudomonas aeruginosa, Escherichia coli and Staphylococcus aureus; it has a strong antibacterial effect on bacteria in the dormant, proliferative and stable phases, and shows significant activity especially against drug-resistant strains.