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Founded in:
2002-05-30 -
Country:
China -
Address:
No. 29, Nanhuan Road, Xinghua City, Jiangsu Province -
Tax NO.:
91321281737808995C -
Registered Funds:
281.58 million yuan -
Website:
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Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Carbazochrome |
It is an oxidized derivative of adrenaline and has no adrenaline-mimetic effect, so it does not affect blood pressure and heart rate, but it can enhance the resistance of capillaries to damage, stabilize the acidic mucopolysaccharides in blood vessels and surrounding tissues, reduce the permeability of capillaries, and enhance the retraction effect of damaged capillary ends, making it difficult for blood clots to fall off the tube wall, thereby shortening the hemostasis time, but it does not affect the coagulation process.
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It is an oxidized derivative of adrenaline and has no adrenaline-mimetic effect, so it does not affect blood pressure and heart rate, but it can enhance the resistance of capillaries to damage, stabilize the acidic mucopolysaccharides in blood vessels and surrounding tissues, reduce the permeability of capillaries, and enhance the retraction effect of damaged capillary ends, making it difficult for blood clots to fall off the tube wall, thereby shortening the hemostasis time, but it does not affect the coagulation process. |
69-81-8 | 4 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Phenytoin sodium |
Antiepileptic drugs, antiarrhythmic drugs. The therapeutic dose does not cause sedation and hypnosis, and has a selective antagonistic effect on the tonic phase of super-strong electric shock and convulsion, but is ineffective or even aggravates the spasm phase. Therefore, it is effective for grand mal epileptic seizures, but ineffective for absence seizures. Its antiepileptic mechanism is to increase the outflow of sodium ions from cells, reduce the inflow of sodium ions, stabilize the nerve cell membrane, increase the excitation threshold, and reduce the spread of high-frequency discharges in the lesion. In addition, it shortens the action potential interval and the effective refractory period, and can also inhibit the influx of calcium ions, reduce myocardial automaticity, inhibit the sympathetic center, and have an inhibitory effect on the ectopic rhythm points of the atria and ventricles, and increase the thresholds for atrial fibrillation and ventricular fibrillation.
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Antiepileptic drugs, antiarrhythmic drugs. The therapeutic dose does not cause sedation and hypnosis, and has a selective antagonistic effect on the tonic phase of super-strong electric shock and convulsion, but is ineffective or even aggravates the spasm phase. Therefore, it is effective for grand mal epileptic seizures, but ineffective for absence seizures. Its antiepileptic mechanism is to increase the outflow of sodium ions from cells, reduce the inflow of sodium ions, stabilize the nerve cell membrane, increase the excitation threshold, and reduce the spread of high-frequency discharges in the lesion. In addition, it shortens the action potential interval and the effective refractory period, and can also inhibit the influx of calcium ions, reduce myocardial automaticity, inhibit the sympathetic center, and have an inhibitory effect on the ectopic rhythm points of the atria and ventricles, and increase the thresholds for atrial fibrillation and ventricular fibrillation. |
630-93-3 | 14 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Lincomycin hydrochloride |
It has high antibacterial activity against common aerobic Gram-positive bacteria, such as Staphylococcus aureus (including those resistant to penicillin G), Staphylococcus epidermidis, beta-hemolytic Streptococcus, viridans Streptococcus and Streptococcus pneumoniae. It has good antibacterial effect on anaerobic bacteria, including Clostridium diphtheriae, and Clostridium perfringens. It has no activity against Gram-negative bacteria such as enterococci, meningococci, Neisseria gonorrhoeae, and Haemophilus influenzae, as well as fungi. There is no cross-resistance between this product and penicillin, chloramphenicol, cephalosporins and tetracyclines, and there is partial cross-resistance with macrolides. This product acts on the 50S subunit of the ribosome of sensitive bacteria, preventing the extension of the peptide chain, thereby inhibiting the protein synthesis of bacterial cells. It is generally an antibacterial agent, but at high concentrations, it also has a bactericidal effect on certain bacteria.
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It has high antibacterial activity against common aerobic Gram-positive bacteria, such as Staphylococcus aureus (including those resistant to penicillin G), Staphylococcus epidermidis, beta-hemolytic Streptococcus, viridans Streptococcus and Streptococcus pneumoniae. It has good antibacterial effect on anaerobic bacteria, including Clostridium diphtheriae, and Clostridium perfringens. It has no activity against Gram-negative bacteria such as enterococci, meningococci, Neisseria gonorrhoeae, and Haemophilus influenzae, as well as fungi. There is no cross-resistance between this product and penicillin, chloramphenicol, cephalosporins and tetracyclines, and there is partial cross-resistance with macrolides. This product acts on the 50S subunit of the ribosome of sensitive bacteria, preventing the extension of the peptide chain, thereby inhibiting the protein synthesis of bacterial cells. It is generally an antibacterial agent, but at high concentrations, it also has a bactericidal effect on certain bacteria. |
859-18-7 | 30 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Carbazochrome |
It is an oxidized derivative of adrenaline and has no adrenaline-mimetic effect, so it does not affect blood pressure and heart rate, but it can enhance the resistance of capillaries to damage, stabilize the acidic mucopolysaccharides in blood vessels and surrounding tissues, reduce the permeability of capillaries, and enhance the retraction effect of damaged capillary ends, making it difficult for blood clots to fall off the tube wall, thereby shortening the hemostasis time, but it does not affect the coagulation process.
More
It is an oxidized derivative of adrenaline and has no adrenaline-mimetic effect, so it does not affect blood pressure and heart rate, but it can enhance the resistance of capillaries to damage, stabilize the acidic mucopolysaccharides in blood vessels and surrounding tissues, reduce the permeability of capillaries, and enhance the retraction effect of damaged capillary ends, making it difficult for blood clots to fall off the tube wall, thereby shortening the hemostasis time, but it does not affect the coagulation process. |
69-81-8 | 4 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Oxytetracycline |
Tetracycline antibiotics. This product is a broad-spectrum antibacterial agent. Many Rickettsia, Mycoplasma, Chlamydia, and Spirochete are sensitive to this product. Enterococci are resistant to it. Others such as Actinomycetes, Bacillus anthracis, Listeria monocytogenes, Clostridium, Nocardia, Vibrio, Brucella, Campylobacter, Yersinia, etc. are also sensitive to this product. Due to the widespread use of oxytetracycline and tetracyclines over the years, common clinical pathogens have serious resistance to oxytetracycline, including Gram-positive bacteria such as Staphylococcus and most Gram-negative bacteria. There is cross-resistance between this product and different varieties of tetracycline antibiotics. The mechanism of action of this product is that the drug can specifically bind to the A position of the 30S subunit of the bacterial ribosome, inhibit the growth of the peptide chain and affect the synthesis of bacterial proteins.
More
Tetracycline antibiotics. This product is a broad-spectrum antibacterial agent. Many Rickettsia, Mycoplasma, Chlamydia, and Spirochete are sensitive to this product. Enterococci are resistant to it. Others such as Actinomycetes, Bacillus anthracis, Listeria monocytogenes, Clostridium, Nocardia, Vibrio, Brucella, Campylobacter, Yersinia, etc. are also sensitive to this product. Due to the widespread use of oxytetracycline and tetracyclines over the years, common clinical pathogens have serious resistance to oxytetracycline, including Gram-positive bacteria such as Staphylococcus and most Gram-negative bacteria. There is cross-resistance between this product and different varieties of tetracycline antibiotics. The mechanism of action of this product is that the drug can specifically bind to the A position of the 30S subunit of the bacterial ribosome, inhibit the growth of the peptide chain and affect the synthesis of bacterial proteins. |
79-57-2 | 34 |