It is a multi-component macrolide antibiotic produced by Streptomyces. Its antibacterial properties are similar to those of erythromycin. It has strong antibacterial effects on Gram-positive bacteria and mycoplasmas, and also has antibacterial effects on some Gram-negative bacteria such as Neisseria meningitidis and Neisseria gonorrhoeae. It works by acting on the 50S subunit of bacterial ribosomes and inhibiting the synthesis of bacterial proteins. It is a growth-stage antibacterial drug.

It is a multi-component macrolide antibiotic produced by Streptomyces. Its antibacterial properties are similar to those of erythromycin. It has strong antibacterial effects on Gram-positive bacteria and mycoplasmas, and also has antibacterial effects on some Gram-negative bacteria such as Neisseria meningitidis and Neisseria gonorrhoeae. It works by acting on the 50S subunit of bacterial ribosomes and inhibiting the synthesis of bacterial proteins. It is a growth-stage antibacterial drug.

This product is a gastrin receptor antagonist, which can significantly inhibit the secretion of gastric acid and pepsin caused by gastrin, but has no obvious effect on the gastric acid secretion caused by histamine and vagus nerve stimulation. It can increase the content of hexosamine in the gastric mucosa, promote glycoprotein synthesis, protect the gastric mucosa and promote healing, improve the symptoms of peptic ulcer and promote ulcer healing. The use of this product to treat peptic ulcer and gastritis does not cause rebound of gastric acid secretion, and gastric acid secretion can remain at a normal level for up to half a year after the treatment is terminated. In addition, this product has a choleretic effect, preventing stone formation by stimulating bile acid-independent bile secretion, changing the stone-forming factors in bile, and antagonizing CCK.

This product has a broad-spectrum antibacterial effect, especially high antibacterial activity against aerobic Gram-negative bacilli. It has good antibacterial activity in vitro against the following bacteria: most bacteria of the Enterobacteriaceae family, including Citrobacter, cloacae, Enterobacter aerogenes, Escherichia coli, Klebsiella, Proteus, Salmonella, Shigella, Vibrio, Yersinia, etc. It often has antibacterial activity against multi-drug resistant bacteria. It has high antibacterial activity against penicillin-resistant Neisseria gonorrhoeae, enzyme-producing Haemophilus influenzae and Moraxella. It has antibacterial activity against most strains of Pseudomonas such as Pseudomonas aeruginosa. This product has antibacterial activity against methicillin-sensitive Staphylococcus aureus, and only moderate antibacterial activity against Streptococcus pneumoniae, hemolytic Streptococcus and Enterococcus faecalis. It also has good effects on Chlamydia trachomatis, Mycoplasma, Legionella, and antibacterial activity against Mycobacterium tuberculosis and atypical mycobacteria. Poor antibacterial activity against anaerobic bacteria. Ciprofloxacin is a bactericide that acts on the A subunit of bacterial DNA gyrase, inhibiting DNA synthesis and replication, leading to bacterial death.

Increase the "glycosamine" content of gastric mucosal cells, improve the defense of gastric mucosa, prolong the life of gastric epithelial cells, and accelerate the healing of ulcers

Promote mucosal regeneration and accelerate ulcer healing

1. Anti-inflammatory effects: (1) Anti-allergic effects, including inhibition of local allergic necrosis and Schwartzman phenomenon in rabbits; enhancing the inhibitory effect of corticosteroids on stress response, and antagonizing the anti-granulation and thymic atrophy effects of hormones. (2) Inhibitory effect on arachidonic acid metabolic enzymes, selectively inhibiting the phosphorylation of phospholipase A2 and lipoxygenase and inhibiting their activation. 2. Immunomodulatory effects: (1) Regulatory effect on T cell activation; (2) Inducing effect on g interferon; (3) Activating NK cells; (4) Promoting the differentiation of extrathymic T lymphocytes. 3. Inhibitory effect on experimental hepatocyte damage: In vitro cultured rat hepatocyte cell line, inhibited hepatocyte damage caused by carbon tetrachloride. 4. Inhibitory effect on virus proliferation and inactivation: Prolonged the survival days of mice infected with hepatitis virus, prevented the occurrence of vaccinia virus in rabbits, and in vitro experiments observed the inhibition of the proliferation of herpes virus and the inactivation of viruses.