This product is a broad-spectrum antibacterial agent. Its mechanism of action is to compete with para-aminobenzoic acid (PABA) in the body, inhibit dihydrofolate synthase, and thus hinder the growth and reproduction of bacteria.

This product is a broad-spectrum antibacterial agent. Its mechanism of action is to compete with para-aminobenzoic acid (PABA) in the body, inhibit dihydrofolate synthase, and thus hinder the growth and reproduction of bacteria.

This product is a broad-spectrum antibacterial agent. Its mechanism of action is to compete with para-aminobenzoic acid (PABA) in the body, inhibit dihydrofolate synthase, and thus hinder the growth and reproduction of bacteria.

This product is a broad-spectrum antibacterial agent. Its mechanism of action is to compete with para-aminobenzoic acid (PABA) in the body, inhibit dihydrofolate synthase, and thus hinder the growth and reproduction of bacteria.

This product is a broad-spectrum antibacterial agent. Its mechanism of action is to compete with para-aminobenzoic acid (PABA) in the body, inhibit dihydrofolate synthase, and thus hinder the growth and reproduction of bacteria.

This product is a broad-spectrum antibacterial agent. Its mechanism of action is to compete with para-aminobenzoic acid (PABA) in the body, inhibit dihydrofolate synthase, and thus hinder the growth and reproduction of bacteria.

Diclofenac sodium is a non-steroidal anti-inflammatory analgesic derived from phenylacetic acid. Its mechanism of action is to inhibit the activity of cyclooxygenase, thereby blocking the conversion of arachidonic acid to prostaglandins. At the same time, it can also promote the combination of arachidonic acid with triglycerides, reduce the concentration of free arachidonic acid in cells, and indirectly inhibit the synthesis of leukotrienes. Animal experiments have confirmed that prostaglandins are one of the mediators of intraocular inflammation, which can lead to the collapse of the blood-aqueous barrier, vasodilation, increased vascular permeability, leukocyte chemotaxis, non-cholinergic mechanism pupil constriction, etc. Diclofenac sodium is a stronger non-steroidal anti-inflammatory drug. Its inhibitory effect on prostaglandin synthesis is stronger than that of aspirin and indomethacin. Diclofenac sodium eye drops have a protective effect on the destruction of the blood-aqueous barrier caused by mechanical, chemical, biological and other stimuli.

This product can selectively inhibit the microRNA virus polymerase of infected cells, inhibiting the RNA polymerase of the virus coding RNA in the infected cells, thereby inhibiting the synthesis of viral RNA, thereby inhibiting the virus. 50mu;g/ml can effectively inhibit various strains of human enterovirus, Coxsackie virus and polio virus; 10mu;g/ml can inhibit acute epidemic hemorrhagic tuberculosis and keratitis.

It has high antibacterial activity against common aerobic Gram-positive bacteria, such as Staphylococcus aureus (including those resistant to penicillin G), Staphylococcus epidermidis, β-hemolytic Streptococcus, viridans Streptococcus and Streptococcus pneumoniae. It has good antibacterial effect against anaerobic bacteria, including Clostridium diphtheriae, and Clostridium perfringens. It has no activity against Gram-negative bacteria such as Enterococcus, meningococci, Neisseria gonorrhoeae, and Haemophilus influenzae, as well as fungi. It has no cross-resistance with penicillin, chloramphenicol, cephalosporins and tetracyclines, but has partial cross-resistance with macrolides. It acts on the 50S subunit of the ribosome of sensitive bacteria, preventing the extension of the peptide chain, thereby inhibiting the protein synthesis of bacterial cells. It is generally an antibacterial agent, but at high concentrations, it also has a bactericidal effect on certain bacteria.

Azithromycin is an azalide antibiotic, and its mechanism of action is to interfere with the synthesis of proteins (without affecting the synthesis of nucleic acids) by binding to the 50s ribosome subunits of sensitive microorganisms. It is effective against a variety of pathogenic bacteria, including Gram-positive aerobic microorganisms (such as Staphylococcus aureus, Streptococcus pyogenes, etc.), Gram-negative aerobic microorganisms (such as Haemophilus influenzae, Moraxella catarrhalis, etc.), and avian intracellular mycobacterium complex.