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Yingzi Maishitong (Hebi) Pharmaceutical Co., Ltd.
  • Founded in:

    2003-10-09
  • Country:

    China China
  • Address:

    Haihe Road, Hebi Economic and Technological Development Zone
  • Tax NO.:

    914106007538916455
  • Registered Funds:

    11.78 million yuan
  • Email:

Related Drugs
Compound diphenoxylate tablets
This product is a compound preparation. Its main ingredient is diphenoxylate.
Name Description Content CAS NO. Registered Holders
Diphenoxylate

No specific description of pharmacological action is provided

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No specific description of pharmacological action is provided

0
Busulfan Tablets
Busulfan
Name Description Content CAS NO. Registered Holders
Busulfan

It is a bifunctional alkylating agent belonging to the class of bismethyl sulfonates and is a non-specific drug for the cell cycle. When it enters the human body, the ring structure of the sulfonate group opens and destroys the structure and function of DNA by alkylating guanine in the cell's DNA. The cytotoxic effect of this product is almost entirely manifested in the inhibition of hematopoietic function, mainly manifested in the obvious inhibition of granulocyte production. The second is the inhibition of platelets and red blood cells, and the inhibition of lymphocytes is very weak.

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It is a bifunctional alkylating agent belonging to the class of bismethyl sulfonates and is a non-specific drug for the cell cycle. When it enters the human body, the ring structure of the sulfonate group opens and destroys the structure and function of DNA by alkylating guanine in the cell's DNA. The cytotoxic effect of this product is almost entirely manifested in the inhibition of hematopoietic function, mainly manifested in the obvious inhibition of granulocyte production. The second is the inhibition of platelets and red blood cells, and the inhibition of lymphocytes is very weak.

0
Norcantharidin tablets
The main ingredient of this product is norcantharidin, and its chemical name is: exo-1:2-cis-3:6-oxo-hexahydrophthalic anhydride. Molecular formula: C8H8O4 Molecular weight: 168.15
Name Description Content CAS NO. Registered Holders
Norcantharidin

It has a destructive or inhibitory effect on the morphology or proliferation of liver cancer BEL-7402, esophageal cancer CaEs-17, laryngeal cancer SMMC-7721, cervical cancer Hela and other cell lines; it has a certain inhibitory effect on mouse Ehrlich ascites cancer, sarcoma 180 and solid liver cancer, and can improve the respiratory control rate and lysosomal enzyme activity of mouse ascites liver cancer H22 tumor cell microsomes, inhibit cancer cell DNA synthesis, and destroy cancer cell skeleton and ultrastructure. It interferes with cancer cell division, blocks it in the M phase, and affects its cycle speed, destroys the cancer cell skeleton (microfilaments, microtubules), affects the ultrastructure of cancer cells, causes damage to mitochondria, microvilli and plasma membrane, blocks the M phase of the cell division cycle, and interferes with cancer cell division; the LD50 of mouse gavage cells is 43.3mg/Kg. After taking the drug, it can be seen that the white blood cells have a certain degree of increase, improve liver function and inhibit the replication of hepatitis B virus. It can be used in combination with other chemotherapy drugs to improve the efficacy and reduce side effects.

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It has a destructive or inhibitory effect on the morphology or proliferation of liver cancer BEL-7402, esophageal cancer CaEs-17, laryngeal cancer SMMC-7721, cervical cancer Hela and other cell lines; it has a certain inhibitory effect on mouse Ehrlich ascites cancer, sarcoma 180 and solid liver cancer, and can improve the respiratory control rate and lysosomal enzyme activity of mouse ascites liver cancer H22 tumor cell microsomes, inhibit cancer cell DNA synthesis, and destroy cancer cell skeleton and ultrastructure. It interferes with cancer cell division, blocks it in the M phase, and affects its cycle speed, destroys the cancer cell skeleton (microfilaments, microtubules), affects the ultrastructure of cancer cells, causes damage to mitochondria, microvilli and plasma membrane, blocks the M phase of the cell division cycle, and interferes with cancer cell division; the LD50 of mouse gavage cells is 43.3mg/Kg. After taking the drug, it can be seen that the white blood cells have a certain degree of increase, improve liver function and inhibit the replication of hepatitis B virus. It can be used in combination with other chemotherapy drugs to improve the efficacy and reduce side effects.

5442-12-6 3
Gastric flat film
This product is a compound preparation. Each tablet contains the main ingredients: 0.0125 grams of magnesium trisilicate, 0.05 grams of aluminum hydroxide, and 0.25 grams of alginate.
Name Description Content CAS NO. Registered Holders
Magnesium trisilicate

This product can neutralize gastric acid and protect the gastric mucosa with a long-lasting effect.

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This product can neutralize gastric acid and protect the gastric mucosa with a long-lasting effect.

0.0125g 14987-04-3 11
Aluminum hydroxide

This product can neutralize gastric acid and protect the gastric mucosa with a long-lasting effect.

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This product can neutralize gastric acid and protect the gastric mucosa with a long-lasting effect.

0.05g 21645-51-2 14
Alginic acid

This product can neutralize gastric acid and protect the gastric mucosa with a long-lasting effect.

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This product can neutralize gastric acid and protect the gastric mucosa with a long-lasting effect.

0.25g 9005-32-7 4
Propafenone Hydrochloride Tablets
The main ingredient and chemical name of this product are: 3-phenyl-1-[2-[3-(propylamino)-2-hydroxypropoxy]-phenyl]-1-propanone hydrochloride
Name Description Content CAS NO. Registered Holders
Propafenone Hydrochloride

This product belongs to the class Ic (i.e., directly acting on the cell membrane) antiarrhythmic drug. The experimental results of isolated animal myocardium indicate that 0.5-1ug/min can reduce the depolarization effect during contraction, thereby prolonging conduction, slightly prolonging the duration of action potential and the effective refractory period, and can increase the threshold potential of myocardial cells, significantly reducing the spontaneous excitability of the myocardium. It acts on both the atrium and ventricle (mainly affecting the Purkinje fibers, with a smaller effect on the myocardium), and also on the formation and conduction of excitement. Clinical data show that the therapeutic dose (300mg orally and 30mg intravenously) can reduce the irritability of the myocardium, the effect is long-lasting, PQ and QRS are increased, and the effective refractory period of the atrium and atrioventricular node is prolonged. It has an antagonistic effect on various types of experimental arrhythmias.

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This product belongs to the class Ic (i.e., directly acting on the cell membrane) antiarrhythmic drug. The experimental results of isolated animal myocardium indicate that 0.5-1ug/min can reduce the depolarization effect during contraction, thereby prolonging conduction, slightly prolonging the duration of action potential and the effective refractory period, and can increase the threshold potential of myocardial cells, significantly reducing the spontaneous excitability of the myocardium. It acts on both the atrium and ventricle (mainly affecting the Purkinje fibers, with a smaller effect on the myocardium), and also on the formation and conduction of excitement. Clinical data show that the therapeutic dose (300mg orally and 30mg intravenously) can reduce the irritability of the myocardium, the effect is long-lasting, PQ and QRS are increased, and the effective refractory period of the atrium and atrioventricular node is prolonged. It has an antagonistic effect on various types of experimental arrhythmias.

34183-22-7 26
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