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Founded in:
1989-03-31 -
Country:
China -
Address:
No. 41, Shuanghe North Road, Tuoketuo County, Hohhot City, Inner Mongolia Autonomous Region -
Tax NO.:
911501221143634247 -
Registered Funds:
15 million yuan -
Email:
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Aluminum hydroxide |
Antacids, which neutralize stomach acid and protect the ulcer surface
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Antacids, which neutralize stomach acid and protect the ulcer surface |
140mg | 21645-51-2 | 14 |
| Vitamin U (iodomethylmethionine) |
Promote granulation development and mucosal regeneration
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Promote granulation development and mucosal regeneration |
50mg | 0 | |
| Atropa Belladonnal |
Inhibit glandular secretion and relieve pain caused by smooth spasm
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Inhibit glandular secretion and relieve pain caused by smooth spasm |
10mg | 0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| STEPHANIAE TETRANDRAE RADIX |
|
0 | ||
| Tetrapanax papyriferus |
|
0 | ||
| Cmnamomi Mmulus |
|
0 | ||
| TURMERIC 1G [R] |
|
0 | ||
| Calcium sulfate dihydrate |
|
10101-41-4 | 0 | |
| COICIS SEMEN |
|
0 | ||
| Chaenomelis Fructus |
|
0 | ||
| Bark of Oriental Variegated Coralbean |
|
0 | ||
| Caulis lonicerae |
|
0 | ||
| Phellodendri Chinensis Cortex |
|
0 | ||
| Talc |
|
14807-96-6 | 26 | |
| Forsythiae Fructus |
|
0 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Calcium lactate |
Participates in bone formation and reconstruction of bone tissue after fracture, as well as muscle contraction, nerve transmission, coagulation mechanism, and can reduce the permeability of capillaries, etc.
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Participates in bone formation and reconstruction of bone tissue after fracture, as well as muscle contraction, nerve transmission, coagulation mechanism, and can reduce the permeability of capillaries, etc. |
0.05g | 814-80-2 | 10 |
| CALCIUM GLUCONATE MONOHYDRATE |
Participates in bone formation and reconstruction of bone tissue after fracture, as well as muscle contraction, nerve transmission, coagulation mechanism, and can reduce the permeability of capillaries, etc.
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Participates in bone formation and reconstruction of bone tissue after fracture, as well as muscle contraction, nerve transmission, coagulation mechanism, and can reduce the permeability of capillaries, etc. |
0.05g | 66905-23-5 | 33 |
| Calcium phosphate dibasic |
Participates in bone formation and reconstruction of bone tissue after fracture, as well as muscle contraction, nerve transmission, coagulation mechanism, and can reduce the permeability of capillaries, etc.
More
Participates in bone formation and reconstruction of bone tissue after fracture, as well as muscle contraction, nerve transmission, coagulation mechanism, and can reduce the permeability of capillaries, etc. |
0.05g | 7757-93-9 | 14 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| Chloramphenicol |
This product has a broad-spectrum antimicrobial effect in vitro, including aerobic Gram-negative and Gram-positive bacteria, anaerobes, Rickettsia, spirochetes and Chlamydia. It has bactericidal effects on the following bacteria: Haemophilus influenzae, Streptococcus pneumoniae and Neisseria meningitidis. It only has antibacterial effects on the following bacteria: Staphylococcus aureus, Streptococcus pyogenes, Streptococcus viridans, Group B hemolytic Streptococcus, Escherichia coli, Klebsiella pneumoniae, Proteus mirabilis, Salmonella typhi, Salmonella paratyphi, Shigella, Bacteroides fragilis and other anaerobic bacteria. The following bacteria are usually resistant to chloramphenicol: Pseudomonas aeruginosa, Acinetobacter, Enterobacter, Serratia marcescens, indole-positive Proteus, methicillin-resistant Staphylococcus and Enterococcus. This product is an antibacterial agent. Chloramphenicol is fat-soluble and its mechanism of action is to diffuse into bacterial cells and reversibly bind to the 50S subunit of bacterial ribosomes, thereby hindering the growth of peptide chains (possibly due to the inhibition of transpeptidase), thereby inhibiting the formation of peptide chains and preventing protein synthesis.
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This product has a broad-spectrum antimicrobial effect in vitro, including aerobic Gram-negative and Gram-positive bacteria, anaerobes, Rickettsia, spirochetes and Chlamydia. It has bactericidal effects on the following bacteria: Haemophilus influenzae, Streptococcus pneumoniae and Neisseria meningitidis. It only has antibacterial effects on the following bacteria: Staphylococcus aureus, Streptococcus pyogenes, Streptococcus viridans, Group B hemolytic Streptococcus, Escherichia coli, Klebsiella pneumoniae, Proteus mirabilis, Salmonella typhi, Salmonella paratyphi, Shigella, Bacteroides fragilis and other anaerobic bacteria. The following bacteria are usually resistant to chloramphenicol: Pseudomonas aeruginosa, Acinetobacter, Enterobacter, Serratia marcescens, indole-positive Proteus, methicillin-resistant Staphylococcus and Enterococcus. This product is an antibacterial agent. Chloramphenicol is fat-soluble and its mechanism of action is to diffuse into bacterial cells and reversibly bind to the 50S subunit of bacterial ribosomes, thereby hindering the growth of peptide chains (possibly due to the inhibition of transpeptidase), thereby inhibiting the formation of peptide chains and preventing protein synthesis. |
56-75-7 | 14 |
| Name | Description | Content | CAS NO. | Registered Holders |
|---|---|---|---|---|
| 1-(2,6-Dichlorophenyl)-2-indolinone |
Diclofenac is a non-steroidal anti-inflammatory analgesic derived from phenylacetic acid. Its mechanism of action is to inhibit the activity of cyclooxygenase, thereby blocking the conversion of arachidonic acid into prostaglandins. At the same time, it can also promote the combination of arachidonic acid with triglycerides (triacylglycerols), reduce the concentration of free arachidonic acid in cells, and indirectly inhibit the synthesis of leukotrienes. Diclofenac is a stronger non-steroidal anti-inflammatory drug, and its inhibitory effect on prostaglandin synthesis is stronger than that of aspirin and indomethacin.
More
Diclofenac is a non-steroidal anti-inflammatory analgesic derived from phenylacetic acid. Its mechanism of action is to inhibit the activity of cyclooxygenase, thereby blocking the conversion of arachidonic acid into prostaglandins. At the same time, it can also promote the combination of arachidonic acid with triglycerides (triacylglycerols), reduce the concentration of free arachidonic acid in cells, and indirectly inhibit the synthesis of leukotrienes. Diclofenac is a stronger non-steroidal anti-inflammatory drug, and its inhibitory effect on prostaglandin synthesis is stronger than that of aspirin and indomethacin. |
15307-86-5 | 4 |