This product has the physical and chemical characteristics of strong negative charge, which can interfere with many links of the blood coagulation process and has anticoagulant effect in vivo and in vitro. Its mechanism of action is relatively complex, mainly through binding with antithrombin III (AT-III), thereby enhancing the latter's inhibitory effect on activated coagulation factors II, IX, X, XI and XII. Its consequences involve preventing platelet aggregation and destruction, hindering the formation of coagulation activating enzymes; preventing prothrombin from becoming thrombin; inhibiting thrombin, thereby preventing fibrinogen from becoming fibrin.

It has a significant effect on increasing the number of peripheral blood leukocytes in normal rabbits. The peak of the leukocyte count occurs on the 4th day after the first administration, which is 72% higher than before the administration, which is better than the leukocyte-raising drug shark liver alcohol. It has a significant effect on increasing leukocytes in rabbits induced by cobalt 60-gamma rays and deep X-ray irradiation. It also has a preventive effect on leukopenia when administered two days before irradiation. It can also prevent leukopenia in mice caused by mitomycin C. It has a synergistic inhibitory effect on solid Ehrlich carcinoma when used in combination with cyclophosphamide, and the toxicity of leukopenia caused by cyclophosphamide is significantly reduced.

In acute toxicity experiments, the LD50 in mice and rats was greater than 4000 mg/Kg. In subacute toxicity experiments, Wistar male and female rats were given 40 mg, 200 mg and 1000 mg/Kg per day for 5 weeks, and male and female rabbits were given 6 mg, 60 mg and 600 mg/Kg per day for 23 consecutive days. There were no special changes in the blood, urine and morphological observations of the test animals. In chronic toxicity experiments, Wistar male and female rats were given 6 mg, 60 mg and 600 mg/Kg per day for 26 consecutive weeks. There were no special changes in the general state, blood and urine examinations, and morphological observations of the test animals.

It can stimulate the uterus, improve microcirculation, resist myocardial ischemia, reduce blood viscosity, resist thrombosis, anti-inflammatory and other effects (such as lowering blood pressure, stimulating the respiratory center, diuretic and curare-like effects).

This product has the effects of stimulating the uterus, improving microcirculation, resisting myocardial ischemia, and reducing blood viscosity. 1. Motherwort paste has a direct stimulating effect on the isolated uterus of rabbits, guinea pigs, and dogs. Its effect is similar to that of posterior pituitary hormone, but weaker. The stimulating effect of motherwort alkaloids on the uterus can last for several hours, but it can be restored after flushing. 2. Anti-thrombosis: Motherwort inhibits thrombosis by inhibiting platelet function, inhibiting the internal and external coagulation systems, and promoting fibrinolytic activity. It also has a certain effect of promoting dissolution. Experiments have shown that it has an anti-thrombotic effect both in vivo and in vitro. 3. Anti-myocardial ischemia: Motherwort has a significant increase in coronary blood flow and a considerable slowing effect on the myocardial ischemia model of isolated guinea pigs. It can show effects similar to alpha and beta receptor blockers. It has the effects of increasing coronary flow, reducing coronary resistance, slowing heart rate and reducing cardiac output. It can also reduce the scope of myocardial infarction. 4. Improve microcirculation: Motherwort can change the microblood flow of rats with mesenteric microcirculation disorders from granular to linear, and reopen the closed capillaries. Increase the blood flow of liver tissue in anesthetized rats. Increase the blood flow of femoral arteries in anesthetized dogs. 5. Reduce blood viscosity: Motherwort can reduce blood flow and plasma viscosity, reduce the red blood cell aggregation index and K (red blood cell hardness index) value. It can significantly reduce the low shear viscosity and high shear viscosity of whole blood. In vitro experiments also show that motherwort has a significant effect in reducing blood viscosity. 6. Anti-inflammatory: Its anti-inflammatory activity is mainly due to progesterone compounds. 7. Others: Motherwort also has antihypertensive, respiratory center stimulating, diuretic and curare-like effects.

This product is a coenzyme. Adenosine triphosphate disodium is a nucleotide derivative that participates in the metabolism of fat, protein, sugar, nucleic acid and nucleotide in the body. When energy is needed for absorption, secretion, muscle contraction and biochemical synthesis reactions in the body, adenosine triphosphate is decomposed into adenosine diphosphate and phosphate groups, and energy is released at the same time. Adenosine triphosphate disodium can penetrate the blood-cerebrospinal fluid barrier, improve the stability and reconstruction ability of the membrane structure of nerve cells, and promote the regrowth of neurites.