It has a layered structure and non-uniform charge distribution, and has a fixation and inhibition effect on viruses, bacteria and toxins produced in the digestive tract; it has the ability to cover the digestive tract mucosa, and through interaction and binding with mucus glycoproteins, it repairs and improves the defense function of the mucosal barrier against attacking factors in terms of both quality and quantity. It does not enter the blood circulation, and is excreted from the body along with the fixed attacking factors with the peristalsis of the digestive tract itself. It does not affect X-ray examinations, does not change the color of stool, and does not change normal intestinal peristalsis.

It achieves antibacterial effect by inhibiting the activity of bacterial DNA gyrase (bacterial topoisomerase II) and hindering bacterial replication. It has the characteristics of broad antibacterial spectrum and strong antibacterial effect, and has good antibacterial effect on most Enterobacteriaceae, Gram-negative bacteria, some Gram-positive bacteria, Legionella, Mycoplasma, Chlamydia, etc. However, it has poor effect on anaerobic bacteria and enterococci.

This product is a broad-spectrum antifungal drug of the acrylamine class. It can specifically interfere with the early biosynthesis of fungal sterols, selectively inhibit the activity of fungal squalene epoxidase, and hinder the squalene epoxidation reaction in the process of fungal cell membrane formation, thereby achieving the effect of killing or inhibiting fungi.

Proton pump inhibitor. This product is a fat-soluble weakly alkaline drug that is easily concentrated in an acidic environment. Therefore, after oral administration, it can be specifically distributed in the secretory tubules of the gastric mucosal parietal cells and converted into the active form of sulfenamide in this high-acid environment. Then, it irreversibly combines with the sulfhydryl group of the H, K-ATPase (also known as the proton pump) in the secretory membrane of the parietal cells through a disulfide bond to form a complex of sulfenamide and the proton pump, thereby inhibiting the activity of the enzyme and blocking the final step of gastric acid secretion. Therefore, this product has a strong and lasting inhibitory effect on gastric acid secretion caused by various reasons.

It is a vasodilator that can dilate coronary arteries and cerebral blood vessels, reduce myocardial oxygen consumption, improve microcirculation and resist platelet aggregation; it can relax smooth muscles; it has a certain antihypertensive effect on normal and hypertensive animals; it has a significant coronary artery dilation effect, which can dilate normal and spasmodic coronary arteries, increase coronary blood flow by 40%, and reduce vascular resistance by 29%; it can inhibit thrombin-induced 5-HT release in platelets.