Amino acids are the basic units that make up human proteins and enzymes, and are involved in human metabolism and various physiological functions.

Amino acids are the basic units that make up human proteins and enzymes, and are involved in human metabolism and various physiological functions.

The activity of phenylalanine hydroxylase in infants and young children is low, and they are prone to hyperphenylalaninemia. Therefore, the dosage should be reduced to adapt to the metabolic characteristics of infants and young children.

Amino acids are the basic units that make up human proteins and enzymes, and are involved in human metabolism and various physiological functions.

The synthesis rate of histidine is slow, which can easily lead to hypohistidinemia, so the dosage should be increased to meet the nutritional needs of children.

Amino acids are the basic units that make up human proteins and enzymes, and are involved in human metabolism and various physiological functions.

Amino acids are the basic units that make up human proteins and enzymes, and are involved in human metabolism and various physiological functions.

Amino acids are the basic units that make up human proteins and enzymes, and are involved in human metabolism and various physiological functions.

Increase the amount of tyrosine to adapt to the metabolic characteristics of infants and young children.

Amino acids are the basic units that make up human proteins and enzymes, and are involved in human metabolism and various physiological functions.

Because of the low activity of cystathionase, hypermethioninemia is prone to occur, so the amount of methionine should be reduced to adapt to the metabolic characteristics of infants and young children.

Amino acids are the basic units that make up human proteins and enzymes, and are involved in human metabolism and various physiological functions.

The final metabolite of sodium lactate is sodium bicarbonate, which can correct metabolic acidosis. When hyperkalemia is accompanied by acidosis, sodium lactate can correct acidosis and allow potassium ions to enter cells from the blood and extracellular fluid.

Under normal circumstances, human blood also contains a small amount of lactic acid, which is mainly produced by glucose or glycogenolysis, and comes from muscles, skin, brain and cells. After lactic acid is produced, it may be converted into glycogen or acetic acid, or enter the tricarboxylic acid cycle and be decomposed into water and carbon dioxide. Therefore, the final metabolite of sodium lactate is sodium bicarbonate, which can correct metabolic acidosis. When hyperkalemia is accompanied by acidosis, sodium lactate can correct acidosis and allow potassium ions to enter cells from blood and extracellular fluid. The main organs that degrade lactic acid are liver and kidney. When lactic acid metabolism in the body is abnormal or impaired, the therapeutic effect is poor.

When hyperkalemia is accompanied by acidosis, sodium lactate can correct the acidosis and allow potassium ions to enter the cells from the blood and extracellular fluid.

Under normal circumstances, human blood also contains a small amount of lactic acid, which is mainly produced by glucose or glycogenolysis, and comes from muscles, skin, brain and cells. After lactic acid is produced, it may be converted into glycogen or acetic acid, or enter the tricarboxylic acid cycle and be decomposed into water and carbon dioxide. Therefore, the final metabolite of sodium lactate is sodium bicarbonate, which can correct metabolic acidosis. When hyperkalemia is accompanied by acidosis, sodium lactate can correct acidosis and allow potassium ions to enter cells from blood and extracellular fluid. The main organs that degrade lactic acid are liver and kidney. When lactic acid metabolism in the body is abnormal or impaired, the therapeutic effect is poor.

The final metabolite is sodium bicarbonate, which can correct metabolic acidosis; when hyperkalemia is accompanied by acidosis, it can correct acidosis and allow potassium ions to enter cells from the blood and extracellular fluid.

Under normal circumstances, human blood also contains a small amount of lactic acid, which is mainly produced by glucose or glycogenolysis, and comes from muscles, skin, brain and cells. After lactic acid is produced, it may be converted into glycogen or acetic acid, or enter the tricarboxylic acid cycle and be decomposed into water and carbon dioxide. Therefore, the final metabolite of sodium lactate is sodium bicarbonate, which can correct metabolic acidosis. When hyperkalemia is accompanied by acidosis, sodium lactate can correct acidosis and allow potassium ions to enter cells from blood and extracellular fluid. The main organs that degrade lactic acid are liver and kidney. When lactic acid metabolism in the body is abnormal or impaired, the therapeutic effect is poor.

When hyperkalemia is accompanied by acidosis, sodium lactate can allow potassium ions to enter cells from blood and extracellular fluid.

Under normal circumstances, human blood also contains a small amount of lactic acid, which is mainly produced by glucose or glycogenolysis, and comes from muscles, skin, brain and cells. After lactic acid is produced, it may be converted into glycogen or acetic acid, or enter the tricarboxylic acid cycle and be decomposed into water and carbon dioxide. Therefore, the final metabolite of sodium lactate is sodium bicarbonate, which can correct metabolic acidosis. When hyperkalemia is accompanied by acidosis, sodium lactate can correct acidosis and allow potassium ions to enter cells from blood and extracellular fluid. The main organs that degrade lactic acid are liver and kidney. When lactic acid metabolism in the body is abnormal or impaired, the therapeutic effect is poor.

Correct the imbalance of branched-chain amino acids and aromatic amino acids in plasma to prevent hepatic coma; generate energy for the body to use through metabolism in muscle, fat, heart, brain and other tissues; enhance the deamination effect; promote liver tissue repair and cell regeneration, reduce plasma non-protein nitrogen and urea nitrogen content, and maintain a positive nitrogen balance.

Correct the imbalance of branched-chain amino acids and aromatic amino acids in plasma to prevent hepatic coma; generate energy for the body to use through metabolism in muscle, fat, heart, brain and other tissues; enhance the deamination effect; promote liver tissue repair and cell regeneration, reduce plasma non-protein nitrogen and urea nitrogen content, and maintain a positive nitrogen balance.

Correct the imbalance of branched-chain amino acids and aromatic amino acids in plasma to prevent hepatic coma; generate energy for the body to use through metabolism in muscle, fat, heart, brain and other tissues; enhance the deamination effect; promote liver tissue repair and cell regeneration, reduce plasma non-protein nitrogen and urea nitrogen content, and maintain a positive nitrogen balance.

Strengthen the deamination effect; it is beneficial to liver tissue repair and cell regeneration, reduce plasma non-protein nitrogen and urea nitrogen content, and maintain a positive nitrogen balance.

Strengthen the deamination effect; it is beneficial to liver tissue repair and cell regeneration, reduce plasma non-protein nitrogen and urea nitrogen content, and maintain a positive nitrogen balance.

Strengthen the deamination effect; it is beneficial to liver tissue repair and cell regeneration, reduce plasma non-protein nitrogen and urea nitrogen content, and maintain a positive nitrogen balance.

This product is a quinolone antibacterial drug, which has strong antibacterial effect on Gram-negative bacteria, including Escherichia coli, Klebsiella pneumoniae, Proteus, Salmonella typhi, Salmonella paratyphi, Shigella, Enterobacter cloacae, Enterobacter aerogenes, Citrobacter, Serratia marcescens, Pseudomonas aeruginosa, Neisseria meningitidis, Haemophilus influenzae, Moracata, Legionella pneumophila, Neisseria gonorrhoeae, etc. It also has moderate antibacterial effect on Gram-positive cocci such as Staphylococcus and hemolytic Streptococcus. The mechanism of action of this product is to inhibit the bacterial DNA gyrase and thus kill the bacteria.