Inhibits DNA-dependent RNA polymerase activity in sensitive cells, especially bacterial RNA polymerase, but does not inhibit mammalian enzymes. It has bactericidal activity against both intracellular and extracellular Mycobacterium tuberculosis at therapeutic concentrations. It has bactericidal activity against slowly and discontinuously growing Mycobacterium tuberculosis, and also has significant activity against Neisseria meningitidis. When single-step mutations occur in DNA-dependent RNA polymerase, resistance to rifampicin will occur.

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It has a strong affinity for steroid metabolic enzymes in the liver, hinders the inactivation of cortisol and aldosterone, shows obvious corticosteroid-like effects (such as anti-inflammatory, anti-allergic and protective membrane structure, etc.), and has no obvious corticosteroid-like adverse reactions; promotes monochromatic metabolism, reduces the release of ALT and AST; induces γ-IFN and interleukin II, increases NK cell activity and the OKT4/OKT8 ratio and activates the reticuloendothelial system; inhibits the release of histamine from mast cells; inhibits the formation of cell membrane phospholipase A2 (PL-A2) and prostaglandin E2 (PGE2) and granulomatous reactions; inhibits the production and formation of free radicals and lipid peroxides, and reduces the activity of prolyl hydroxylase; regulates calcium ion channels, and protects lysosomal membranes and mitochondria.

Glycyrrhizic acid monoamine has a strong affinity for steroid metabolic enzymes in the liver, thereby hindering the inactivation of cortisol and aldosterone. After use, it shows obvious corticosteroid-like effects, such as anti-inflammatory effects, anti-allergic effects, and protective membrane structures; there are no obvious corticosteroid-like adverse reactions. This product can promote monochrome metabolism, reduce the release of ALT and AST; induce γ-IFN and interleukin II, increase NK cell activity and OKT4/OKT8 ratio and activate the reticuloendothelial system; inhibit mast cell release of histamine; inhibit the formation of cell membrane phospholipase A2 (PL-A2) and prostaglandin E2 (PGE2) and granulomatous reactions; inhibit the production and formation of free radicals and lipid peroxides, and reduce the activity of proline hydroxylase; regulate calcium ion channels, protect lysosomal membranes and mitochondria

Glycyrrhizic acid monoamine has a strong affinity for steroid metabolic enzymes in the liver, thereby hindering the inactivation of cortisol and aldosterone. After use, it shows obvious corticosteroid-like effects, such as anti-inflammatory effects, anti-allergic effects, and protective membrane structures; there are no obvious corticosteroid-like adverse reactions. This product can promote monochrome metabolism, reduce the release of ALT and AST; induce γ-IFN and interleukin II, increase NK cell activity and OKT4/OKT8 ratio and activate the reticuloendothelial system; inhibit mast cell release of histamine; inhibit the formation of cell membrane phospholipase A2 (PL-A2) and prostaglandin E2 (PGE2) and granulomatous reactions; inhibit the production and formation of free radicals and lipid peroxides, and reduce the activity of proline hydroxylase; regulate calcium ion channels, protect lysosomal membranes and mitochondria

It combines with blood ammonia in the body to form urea and is excreted from the body, which has the effect of reducing blood ammonia and promoting brain metabolism. It may be a central mediator that can enhance the activity of glucose phosphatase and facilitate the recovery of brain cell function.