It has anti-thrombotic effect. Dipyridamole inhibits platelet aggregation, and high concentration (50μg/ml) can inhibit platelet release. The mechanism of action may be: ⑴ inhibiting platelet uptake of adenosine, which is a platelet reaction inhibitor; ⑵ inhibiting phosphodiesterase, increasing cyclic adenosine monophosphate (cAMP) in platelets; ⑶ inhibiting the formation of thromboxane A2 (TXA2), which is a strong agonist of platelet activity; ⑷ enhancing endogenous PGI2. Dipyridamole has a vasodilating effect.

This product is an oxime compound. Its quaternary ammonium group can tend to the cationic site of the inactive phosphorylated cholinesterase that has been combined with the organophosphorus insecticide. Its nucleophilic group can directly combine with the phosphorylated group of cholinesterase and then detach from the cholinesterase together, so that the cholinesterase can be restored to its original state. It regains its vitality. The effect of rejuvenating the aged cholinesterase that has been inhibited by organophosphorus insecticides for more than 36 hours is very poor. It has no resurrection effect on the cholinesterase inhibited by chronic organic insecticide poisoning. This product has a significant effect on the nicotinic symptoms caused by organophosphorus insecticides, but a weak effect on muscarinic symptoms, and no significant effect on central nervous system symptoms.

This product is a vasodilator that directly acts on the smooth muscle of arterioles and capillaries, dilates blood vessels, improves blood rheology, and reduces peripheral vascular resistance. This product can promote glucose to pass through the blood-brain barrier, enhance the use of glucose and oxygen in brain cells, improve brain sugar metabolism and brain function. It also promotes fat metabolism and reduces hyperlipidemia, high cholesterol, and high fibrinogen.

It has an antihistamine effect and can compete with histamine released from tissues for the H1 receptors on effector cells, thereby stopping allergic reactions; it inhibits central nervous system activity and causes sedative and hypnotic effects; it enhances the effects of antitussive drugs; it has anti-vertigo and anti-tremor paralysis effects; it has local anesthetic, antiemetic and anti-M cholinergic effects.

Isoniazid has a highly selective antibacterial effect on all types of Mycobacterium tuberculosis. It is the synthetic antibacterial drug with the strongest bactericidal effect among current anti-tuberculosis drugs, and has almost no effect on other bacteria. It has a strong effect on Mycobacterium tuberculosis in the growth and reproduction stage, but a weaker and slower effect on the dormant stage. Its mechanism of action may be to inhibit the synthesis of mycolic acid in sensitive bacteria and cause the cell wall to rupture.