2S10 SS-31 Large quantity discounts, factory direct sales, purity 99.9%

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I. Stabilize mitochondrial structure, effectively scavenge free radicals from the source (core mechanism)
Specifically bind to cardiolipin in the inner mitochondrial membrane, maintain the integrity of mitochondrial cristae, ensure the normal assembly of the respiratory chain complex, significantly reduce electron leakage, and fundamentally reduce the generation of reactive oxygen species (ROS), with antioxidant efficacy far exceeding that of ordinary antioxidants.
Directly neutralize superoxide anions and hydrogen peroxide within the mitochondria, inhibit the generation of oxidative lipids, reduce the oxidative damage of mitochondrial membranes, prevent the release of apoptotic signals in cells.
Downregulate the TXNIP protein, block the activation of the NLRP3 inflammasome, reduce the release of pro-inflammatory factors such as IL-1β and IL-18, and simultaneously inhibit the systemic low-grade chronic inflammation driven by mitochondria.
Reduce the oxidation loss of cardiolipin, delay mitochondrial aging caused by aging, staying up late, and overexertion, and maintain the complete function of cell mitochondria over the long term.
II. Enhance mitochondrial ATP production, comprehensively improve fatigue and weakness
Optimize the activity of ADP/ATP transport protein (ANT), accelerate energy conversion, significantly increase ATP production in all cells throughout the body, improve morning fatigue, persistent drowsiness during the day, lack of concentration when using the brain, and heaviness throughout the body after prolonged sitting.
Still provide stable energy supply under hypoxic and high-intensity load conditions, alleviate shortness of breath and rapid decline in strength during exercise and physical labor, and extend the endurance of aerobic and strength training.
Improve the persistent physical weakness caused by mitochondrial decline in the elderly, reduce fatigue at rest, increase stable basal energy output, and reduce afternoon drowsiness.
III. Sports recovery, muscle and bone maintenance
Reduce mitochondrial oxidative stress during exercise, significantly reduce the accumulation of lactic acid and oxidative waste, alleviate muscle stiffness after training, delay soreness, shorten the overall repair cycle (PMC).
Protect the mitochondrial of muscle cells, reduce muscle protein breakdown under high-intensity training and calorie deficits, maintain muscle fullness, and delay age-related muscle loss.
Enhance mitochondrial vitality of bone marrow mesenchymal stem cells, promote osteogenic differentiation, enhance bone matrix formation, alleviate limb weakness, joint weakness, and dizziness caused by insufficient oxygen supply to the brain due to aging and overuse, and reduce the rate of bone loss.
Optimize energy supply to joint cartilage cells, alleviate mitochondrial damage in cartilage, relieve joint stiffness and dull pain after prolonged sitting and weight-bearing.
IV. Deep maintenance of cardiovascular and circulatory systems
Protect myocardial mitochondria, improve myocardial energy metabolism, enhance the efficiency of diastole and systole of the heart, alleviate palpitations due to long-term fatigue and high-fat diet, and reduce chest tightness and shortness of breath after exercise.
Repair mitochondrial cells in vascular endothelium, reduce endothelial oxidative damage, maintain vascular elasticity, improve poor peripheral circulation, cold hands and feet, and dizziness caused by insufficient oxygen supply to the brain.
Reduce lipid oxidation deposition in blood vessels, alleviate chronic wear and tear of blood vessels, assist in stabilizing the overall blood flow state.
V. Liver and kidney organ relief and repair
Protect liver cell mitochondria, improve myocardial energy metabolism, enhance the efficiency of diastole and systole of the heart, alleviate palpitations caused by long-term fatigue and high-fat diet.
Repair mitochondrial cells in vascular endothelium, reduce endothelial oxidative damage, maintain vascular elasticity, improve poor peripheral circulation, cold hands and feet, and dizziness caused by insufficient oxygen supply to the brain.
Reduce lipid oxidation deposition in blood vessels, alleviate chronic wear and tear of blood vessels, and assist in stabilizing the overall blood flow state.
VI. Nerve and brain maintenance, improvement of memory and sleep
Protect neuronal mitochondria, reduce abnormal aggregation of α-synuclein, reduce oxidative loss of nerve cells, improve forgetfulness, sluggish response, and cognitive block caused by long-term use of the brain and staying up late.
Relieve sympathetic nerve excitation caused by oxidative stress, calm chaotic thoughts before sleep, reduce brain excitement, shorten sleep duration, and increase the proportion of deep sleep.
Reduce persistent inflammation in the nervous system caused by long-term lack of sleep and high stress, alleviate the state of frequent dreams and waking up still feeling tired, stabilize daytime mood, and reduce irritability and anxiety. VII. Regulation of Glucose Metabolism and Auxiliary Weight Management
Enhance the efficiency of glucose uptake by mitochondria in muscle and fat cells, improve insulin sensitivity, stabilize post-meal blood sugar peaks, and reduce the conversion of sugar into visceral fat accumulation.
Activate the mitochondrial oxidation decomposition pathway in fat cells, increase the proportion of energy supply from resting fat, assist in reducing abdominal visceral fat, and improve metabolic weakness, obesity, and edema conditions.
Inhibit systemic inflammation triggered by blood sugar fluctuations and reduce persistent physical fatigue caused by high-sugar diets.
VIII. Anti-Aging of the Whole Body, Intrinsic Skin Repair and Brightening
Dermal fibroblast mitochondria have sufficient energy, promoting stable synthesis of collagen and elastic fibers, reducing collagen oxidation and degradation, and improving facial sagging, dry lines, and sagging contours.
Remove mitochondrial oxidative free radicals in the skin, reduce skin aging, lack of color, yellowish complexion, sunspots, and inflammatory pigment deposition, and even out the overall skin color.
Reduce chronic inflammation at the skin's base, improve recurrent redness, sensitivity, and rough skin texture, and slow down the aging speed of the skin.
IX. Mitochondrial Protection of the Eyes, Delaying Visual Aging
Protect the mitochondria of retinal cells, reduce photic and oxidative damage to photoreceptor cells, alleviate excessive dryness and fatigue caused by excessive eye use, and delay age-related visual function decline.
Reduce oxidative damage to microvessels at the back of the eye, improve darkening, soreness, and acidification around the eyes caused by long-term screen use and staying up late.

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Handling and Storage

Precautions for safe handling

Handling in a well ventilated place. Wear suitable protective clothing. Avoid contact with skin and eyes. Avoid formation of dust and aerosols. Use non-sparking tools. Prevent fire caused by electrostatic discharge steam.

Conditions for safe storage, including any incompatibilities

Store the container tightly closed in a dry, cool and well-ventilated place. Store apart from foodstuff containers or incompatible materials.