It mainly acts on the dopamine receptors in the presynaptic membrane of the brain to produce an anti-anxiety effect. It has no sedative, muscle relaxant or anticonvulsant effects.

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This product is a derivative of clindamycin. It has no antibacterial activity in vitro. It is hydrolyzed by esterase in vivo to form clindamycin and exert its antibacterial activity. The mechanism of action of clindamycin is to inhibit the synthesis of bacterial proteins. It mainly acts on Gram-positive cocci and anaerobic infections. It has good antibacterial effects on Staphylococcus aureus, Staphylococcus epidermidis, Streptococcus pneumoniae, Streptococcus pyogenes, etc., and is moderately sensitive to Haemophilus influenzae and Neisseria gonorrhoeae. Some erythromycin-resistant Staphylococcus aureus and Staphylococcus epidermidis are still sensitive to this product. It has good antibacterial effects on both Gram-negative and Gram-positive anaerobic bacteria. The MIC50 and MIC90 for Bacteroides fragilis are 0.062 and 0.5 mg/ml respectively, and the MIC50 and MIC90 for Peptostreptococcus are 0.125 and 4 mg/ml respectively.

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This product is a highly selective 5-HT3 receptor antagonist, which has a good preventive and therapeutic effect on nausea and vomiting caused by radiotherapy, chemotherapy and surgery. It inhibits the occurrence of nausea and vomiting by antagonizing the 5-HT3 receptors in the central chemoreceptor area and peripheral vagus nerve endings.