It can prevent the implantation of fertilized eggs and increase the viscosity of cervical mucus, preventing sperm from penetrating

Can inhibit the secretion of gonadotropin, thereby inhibiting ovarian ovulation

This product is an H1 receptor blocker that competitively blocks H1 receptors and produces antihistamine effects. The anti-allergic effect of this product is slightly stronger and more lasting than that of diphenhydramine, and adverse reactions such as drowsiness are less. This product also has a strong antiemetic effect but a weak sedative effect, and has anticholinergic and local anesthetic effects.

The structure is similar to that of papaverine. Single or continuous administration can increase blood flow to the brain, heart, kidneys and limbs. It can directly relax vascular smooth muscle to dilate blood vessels, and has a selective and sustained dilation effect on the brain, kidneys, blood vessels and coronary arteries, thereby increasing blood flow. The effect is weaker and more lasting than that of papaverine. This product can also promote collateral circulation. It has almost no effect on respiration, heart rate, cardiac output, myocardial oxygen consumption, blood pressure, etc.

This product is a gastrin receptor antagonist, which can significantly inhibit the secretion of gastric acid and pepsin caused by gastrin, but has no obvious effect on the gastric acid secretion caused by histamine and vagus nerve stimulation. It can increase the content of hexosamine in the gastric mucosa, promote glycoprotein synthesis, protect the gastric mucosa and promote healing, improve the symptoms of peptic ulcer and promote ulcer healing. The use of this product to treat peptic ulcer and gastritis does not cause rebound of gastric acid secretion, and gastric acid secretion can remain at a normal level for up to half a year after the treatment is terminated. In addition, this product has a choleretic effect: it prevents stone formation by stimulating bile acid-independent bile secretion, changing the stone-forming factors in bile, and antagonizing CCK to inhibit the gallbladder contraction-promoting effect of endogenous CCK.

Acts on dihydrofolate synthase, interfering with the first step of folic acid synthesis; combined with trimethoprim, it can double block the folic acid metabolism of bacteria and has a synergistic antibacterial effect

It acts on the second step of folic acid anabolism and selectively inhibits the action of dihydrofolate reductase; combined with sulfadiazine, it can double block the folic acid metabolism of bacteria and has a synergistic antibacterial effect.

Alkalinize urine to reduce the possibility of urinary crystallization of sulfonamides