When used in combination with ethinyl estradiol, it has a synergistic effect on inhibiting ovulation, causing the endometrium to transform and show secretion, leading to withdrawal bleeding and forming cyclical changes.

It is a long-acting estrogen that inhibits ovarian ovulation by inhibiting the hypothalamus-pituitary-ovarian axis, achieving a long-term contraceptive effect.

This product is a benzimidazole derivative, which is rapidly metabolized into sulfoxide, sulfone alcohol and 2-amine sulfone alcohol in the body. It selectively and irreversibly inhibits the polymerization of the cytoplasmic microtubule system of the parasite's intestinal wall cells, blocks its absorption of various nutrients and glucose, leads to the depletion of endogenous glycogen in the worm, and inhibits the fumarate reductase system, preventing the production of adenosine triphosphate, making it impossible for the worm to survive and reproduce. It can also cause the degeneration of the cytoplasmic microtubules of the worm's intestinal cells, and bind to its microtubule protein, causing intracellular transport blockage, resulting in the accumulation of Golgi endocrine granules, the gradual dissolution of the cytoplasm, and the complete degeneration of the absorbing cells, causing the death of the worm. It has the effect of completely killing hookworm eggs and whipworm eggs and partially killing ascarid eggs. In addition to killing and expelling various nematodes parasitic in animals, it also has a significant killing and expelling effect on tapeworms and cysticerci.

Epalrestat is a reversible non-competitive inhibitor of aldose reductase, which has a selective inhibitory effect on aldose reductase. It can inhibit the accumulation of sorbitol in the red blood cells of patients with diabetic peripheral neuropathy, and improve the patients' subjective symptoms and neurological dysfunction. Animal experiments have shown that epalrestat can significantly inhibit the accumulation of sorbitol in the sciatic nerve, red blood cells, and retina of diabetic model rats, increase motor nerve conduction velocity and autonomic nerve function, improve axonal flow abnormalities, increase the density of myelinated nerve fibers in the sciatic nerve, the thickness of the sural nerve myelin sheath, the axon area, and the axon cylindrical rate, improve the blood flow of the sciatic nerve and restore its inositol content.

It can directly stimulate adrenaline receptors, and can also indirectly stimulate adrenaline receptors by prompting adrenaline nerve endings to release norepinephrine. It has a stimulating effect on both alpha and beta receptors. It can dilate bronchi and constrict local blood vessels, and its action time is longer; it strengthens myocardial contractility, increases cardiac output, and makes venous return to the heart sufficient; it has a stronger central nervous system stimulating effect than adrenaline.

It has ① antihistamine effect, which can compete with histamine released from tissues for H1 receptors on effector cells, thereby stopping allergic reactions; ② at the same time, it inhibits central nervous system activity and causes sedative and hypnotic effects; ③ it enhances the effect of antitussive drugs.

Accelerate the recovery of normal levels of vasoactive substances in the ischemic area of the eye, relieve vasospasm, maintain normal tension and systolic and diastolic function of choroidal vessels, increase blood flow, improve blood supply, and promote rapid recovery of ischemic tissue.

Accelerate the recovery of normal levels of vasoactive substances in the ischemic area of the eye, relieve vasospasm, maintain normal tension and systolic and diastolic function of choroidal vessels, increase blood flow, improve blood supply, and promote rapid recovery of ischemic tissue.