Macrolide antibiotics. It has strong antibacterial effects on Gram-positive bacteria and mycoplasma, and also has antibacterial effects on some Gram-negative bacteria such as meningococci and gonococci.

In the carbon tetrachloride model, it can reduce the serum transaminase and hydroxyproline content in the liver of animals, and reduce the degree of liver lesions; it can reduce the serum DHBV-DNA level of ducks infected with hepatitis B virus (HBV). In vitro experiments show that it can inhibit the proliferation of mouse skin fibroblasts (NIH/3T3 cells) and the synthesis of extracellular matrix; it can inhibit the proliferation and synthesis of extracellular matrix of rat hepatic stellate cells and promote their apoptosis. In the rat liver fibrosis model induced by carbon tetrachloride, dimethylnitrosamine and galactosamine, after oral administration of matrine for prevention and treatment, the serum transaminase, extracellular matrix (hyaluronic acid, laminin, type III collagen and type IV collagen) content of animals, as well as liver cell degeneration, necrosis, inflammatory activity and fibrosis in liver tissue were significantly improved.

Inhibits prostaglandin synthesis and has antipyretic and analgesic effects

Inhibits prostaglandin synthesis and has antipyretic and analgesic effects

Central nervous system stimulants that can enhance the antipyretic and analgesic effects of drugs

Chlortetracycline is a broad-spectrum tetracycline antibiotic that has a good inhibitory effect on Staphylococcus aureus, Streptococcus pyogenes, Pneumococcus, Neisseria gonorrhoeae, and Chlamydia trachomatis. It is suitable for superficial skin infections.

This product is a congener of chloramphenicol. Its antibacterial spectrum and antibacterial effect are similar to those of chloramphenicol. It has a broad-spectrum antimicrobial effect, including aerobic Gram-negative and Gram-positive bacteria, anaerobic bacteria, Rickettsia, spirochetes and Chlamydia. It has a bactericidal effect on Haemophilus influenzae, Streptococcus pneumoniae and Neisseria meningitidis, and only has an antibacterial effect on anaerobic bacteria such as Staphylococcus aureus, Streptococcus pyogenes, Streptococcus viridans, Group B hemolytic Streptococcus, Escherichia coli, Klebsiella pneumoniae, Proteus mirabilis, Salmonella typhi, Salmonella paratyphi, Shigella, Bacteroides fragilis, etc. Pseudomonas aeruginosa, Acinetobacter, Enterobacter, Serratia marcescens, indole-positive Proteus, methicillin-resistant Staphylococcus and Enterococcus are usually resistant to chloramphenicol. This product is an antibacterial agent, which reversibly binds to the 50S subunit of bacterial ribosomes, inhibits peptide chain formation, prevents protein synthesis, and is completely cross-resistant to chloramphenicol. Since thiamphenicol does not bind to glucuronic acid in the liver, it has a high antibacterial activity in vivo. It also has a strong immunosuppressive effect, which is about 6 times stronger than chloramphenicol.